Defining the mechanism(s) by which the cellular inhibitor of apoptosis protein 2 (cIAP2) contributes to early stage atherosclerosis development by directly promoting the participation of key apoptotic pathways within lesion-associated macrophages
Defining the mechanism(s) by which the cellular inhibitor of apoptosis protein 2 (cIAP2) contributes to early stage atherosclerosis development by directly promoting the participation of key apoptotic pathways within lesion-associated macrophages
确定凋亡蛋白细胞抑制剂 2 (cIAP2) 通过直接促进病变相关巨噬细胞内关键凋亡途径的参与来促进早期动脉粥样硬化发展的机制
批准号：
191299
负责人：
Whitman Stewart C
金额：
$48.64万
依托单位国家：
加拿大
项目类别：
Operating Grants
财政年份：
2009
资助国家：
加拿大
项目状态：
已结题
起止时间：
2009-09-01 至 2014-09-01
中文摘要
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英文摘要
Objective: Under different circumstances both anti- and pro-apoptotic mechanisms contribute to the development and progression of atherosclerosis, yet the molecular events leading to these processes are not clearly understood. Macrophages are a key compon
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Apoptosis and early stage atherosclerosis: studies aimed at defining and regulating the mechanism by which cellular inhibitor of apoptosis protein 2 (cIAP2) contributes to early lesion formation
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批准号：179365
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项目类别：Operating Grants
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资助金额：$7.29万
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财政年份：2008
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负责人：Whitman Stewart C
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依托单位：
海外基金
