植物中类受体蛋白激酶识别胞外刺激并激活下游信号通路，在信号转导中发挥着重要作用。尽管数量众多，但只有少数成员功能已知。前期通过基因芯片筛选到富含半胱氨酸的类受体蛋白激酶CRK41定位于质膜且受盐胁迫的诱导表达。基于此，本课题拟对CRK41响应盐胁迫的信号转导功能进行研究，并探究其分子机制。通过研究内容揭示CRK41是否通过微管骨架动态响应盐胁迫；并对其信号通路进行探究，揭示CRK41是否影响MAPK信号途径进而调控微管结合蛋白稳定性，从而诱发微管动态变化。本项目的实施，补充了拟南芥CRK41功能研究的空缺，发现了拟南芥响应盐胁迫的新基因及其信号转导通路，为阐明植物响应盐胁迫的分子机理提供新的理论依据。

In plants, RLKs (receptor-like protein kinases) play essential roles in signal transduction by recognizing extracellular stimuli and activating the downstream signalling pathways. However, there are a lager number of RLK family members whose functions are still unknown. CRK41, a cysteine-rich receptor-like protein kinase screened by gene microarray, is located in the plasma membrane and induced by salt stress in our previous study. Based this interesting phenomenonon, the current project intends to study the signal transduction function of CRK41 in response to salt stress and explore its molecular mechanism. We want to reveal whether CRK41 regulates the dynamics of microtubules in response to salt stress. Furthermore, the signal pathway was explored to unravel whether CRK41 affects the MAPK signal pathway to regulate the stability of microtubule associated proteins, thus inducing the dynamics of microtubules. The project will contribute in filling the lack of functional research of CRK41 in Arabidopsis, and discover a novel gene and signal transduction pathway in response to salt stress. The study will shed new light on our understanding of the molecular regulatory mechanisms during salt stress in plant.