现阶段采用生物、物理或化学等激发子来诱导果实抗病性正成为抑制采后果实病害发生的有效策略。但抗病反应可使果实物质和能量代谢转向于逆境响应，易导致果实品质下降。激发子诱导的敏化反应（Priming）可使植物进入防御准备状态，仅在植物受到病原菌侵染时才表现出己被激活的抗性，因此物质与能量消耗较少。但目前采后果实Priming反应确切的细胞机制及其对果实物质代谢的调控作用仍不清楚。本项目拟以采后跃变型果实水蜜桃和苹果以及非跃变型果实葡萄和草莓果实为试材，以Priming激发子（MeJA、核黄素和BABA）诱导的采后果实Priming反应对果实蔗糖代谢和苯丙烷类代谢的影响为核心研究内容，从解析采后果实Priming 反应细胞机制入手，进而从分子水平阐明采后果实Priming反应与蔗糖代谢和苯丙烷类代谢之间的关联机制并明确诱导抗病对采后果实品质和抗氧化活性的影响，为完善果实贮运保鲜技术提供理论支撑。

Recently, induced resistance by biological, physical or chemical elicitors has become one of the most effective strategies to inhibit postharvest disease in harvested fruits. Nevertheless, the disease resistance can urge the normal metabolism of material and energy turning to stress response, resulting in the quality decline in harvested fruits. The Priming action induced by various elicitors can cause plants into defense preparation state, and the Priming-induced plants exhibit the activated disease resistance only when the plants are infected by pathogens, which therefore consumes less material and energy in plants. However, the specific cellular mechanism and regulation mechanism of metabolism of sucrose and phenylpropnaoid involved in Priming action in harvested fruit are largely unknown. With the typical climacteric fruit peaches and apples and non-climacteric fruit grapes and strawberries as test material, the effects of Priming action in harvested fruit induced by Priming-elicitors such as MeJA, riboflavin and BABA on metabolism of fruit sucrose and phenylpropnaoid are treated as core contents in the present study. Starting with the analysis of cellular mechanism of Priming action in harvested fruit, the present study is to interpret the relation between Priming action and metabolism of fruit sucrose and phenylpropnaoid and to determine the effect of induced disease resistance on fruit quality and antioxidant activity, which lay the foundation for improving fresh-keeping technique in postharvest storage and distribution.