Human Histocompatibility Leukocyte Antigen (HLA)-G Molecules Inhibit NKAT3 Expressing Natural Killer Cells
Human Histocompatibility Leukocyte Antigen (HLA)-G Molecules Inhibit NKAT3 Expressing Natural Killer Cells
复制标题
人类组织相容性白细胞抗原 (HLA)-G 分子抑制 NKAT3 表达的自然杀伤细胞
DOI:
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发表时间:
1997
影响因子:
15.3
通讯作者:
H. Rammensee
中科院分区:
文献类型:
--
作者:
C. Münz;N. Holmes;A. King;Y. W. Loke;M. Colonna;H. Schild;H. Rammensee
The crucial immunological function of the classical human major histocompatibility complex (MHC) class I molecules, human histocompatibility leukocyte antigen (HLA)-A, -B, and -C, is the presentation of peptides to T cells. A secondary function is the inhibition of natural killer (NK) cells, mediated by binding of class I molecules to NK receptors. In contrast, the function of the nonclassical human MHC class I molecules, HLA-E, -F, and -G, is still a mystery. The specific expression of HLA-G in placental trophoblast suggests an important role for this molecule in the immunological interaction between mother and child. The fetus, semiallograft by its genotype, escapes maternal allorecognition by downregulation of HLA-A and HLA-B molecules at this interface. It has been suggested that the maternal NK recognition of this downregulation is balanced by the expression of HLA-G, thus preventing damage to the placenta. Here, we describe the partial inhibition of NK lysis of the MHC class I negative cell line LCL721.221 upon HLA-G transfection. We present three NK lines that are inhibited via the interaction of their NKAT3 receptor with HLA-G and with HLA-Bw4 molecules. Inhibition can be blocked by the anti-NKAT3 antibody 5.133. In conclusion, NK inhibition by HLA-G via NKAT3 may contribute to the survival of the fetal semiallograft in the mother during pregnancy.
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DOI:
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发表时间:
1992
期刊:
Journal of immunology (Baltimore, Md. : 1950)
影响因子:
--
作者:
Zemmour,J;Little,AM;Schendel,DJ;Parham,P
通讯作者:
Parham,P
影响因子:
56.9
作者:
KOVATS, S;MAIN, EK;DEMARS, R
通讯作者:
DEMARS, R
影响因子:
6.4
作者:
Roby,KF;Fei,K;Yang,Y;Hunt,JS
通讯作者:
Hunt,JS
DOI:
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发表时间:
1989
期刊:
Journal of immunology (Baltimore, Md. : 1950)
影响因子:
--
作者:
Shimizu,Y;DeMars,R
通讯作者:
DeMars,R