Response of the bacteriophage T4 replisome to noncoding lesions and regression of a stalled replication fork.
Response of the bacteriophage T4 replisome to noncoding lesions and regression of a stalled replication fork.
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DOI:
10.1016/j.jmb.2010.06.027
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发表时间:
2010-09-03
影响因子:
5.6
通讯作者:
Benkovic SJ
中科院分区:
文献类型:
--
作者:
Nelson SW;Benkovic SJ
DNA is constantly damaged by endogenous and exogenous agents. The resulting DNA lesions have the potential to halt the progression of the replisome, possibly leading to replication fork collapse. Here, we examine the effect of a non-coding DNA lesion in either the leading or lagging strand template on the bacteriophage T4 replisome. A damaged base in the lagging strand template does not affect the progression of the replication fork. Instead, the stalled lagging strand polymerase recycles from the lesion and initiates synthesis of the new Okazaki fragment upstream from the damaged base. In contrast, when the replisome encounters a blocking lesion in the leading strand template, the replication fork only travels approximately 1 kb beyond the point of the DNA lesion before complete replication fork collapse. The primosome and lagging strand polymerase remain active during this period and an Okazaki fragment is synthesized beyond the point of the leading strand lesion. There is no evidence for a new priming event on the leading strand template. Instead, the DNA structure that is produced by the stalled replication fork is a substrate for the DNA repair helicase, UvsW. UvsW catalyzes the regression of a stalled replication fork into a “chicken foot” structure that has been postulated to be an intermediate in an error-free lesion bypass pathway.
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影响因子:
4.8
作者:
McInerney, Peter;O'Donnell, Mike
通讯作者:
O'Donnell, Mike
影响因子:
64.8
作者:
Cox, MM;Goodman, MF;Marians, KJ
通讯作者:
Marians, KJ
影响因子:
4.8
作者:
McInerney, P;O'Donnell, M
通讯作者:
O'Donnell, M
DOI:
10.1073/pnas.121009398
发表时间:
2001-07-17
影响因子:
11.1
作者:
Jones, CE;Mueser, TC;Nossal, NG
通讯作者:
Nossal, NG
影响因子:
4.8
作者:
Kadyrov, FA;Drake, JW
通讯作者:
Drake, JW