The deleterious role of basophils in systemic lupus erythematosus.

The deleterious role of basophils in systemic lupus erythematosus.
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DOI:
10.1016/j.coi.2013.10.003
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发表时间:
2013-12
影响因子:
7
通讯作者:
Charles N
Charles N
中科院分区:
医学2区
文献类型:
--
作者:
Pellefigues C;Charles N

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系统性红斑狼疮是一种多因子起源的复杂自身免疫性疾病。免疫系统的所有部分似乎都受到影响,至少在某种程度上,并有助于疾病的发病机制。由于逃避负选择,自身反应性T和B细胞在SLE患者体内积累,导致自身抗体的产生,主要针对核成分,并随后沉积到靶器官。我们最近发现,嗜碱性细胞以IgE和IL-4依赖的方式,通过扩增自身抗体的产生来促进SLE的发病。在这里,我们总结了我们在小鼠模型和SLE患者中了解到的嗜碱性粒细胞在狼疮中的有害作用。我们讨论了在SLE期间嗜碱性细胞激活和向次级淋巴器官募集的可能途径,以及嗜碱性细胞如何放大自身抗体的产生。
Systemic lupus erythematosus is a complex autoimmune disease of multifactorial origins. All compartments of the immune system appear to be affected, at least in some way, and to contribute to disease pathogenesis. Due to an escape from negative selection autoreactive T and B cells accumulate in SLE patients leading to the production of autoantibodies mainly raised against nuclear components and their subsequent deposition into target organs. We recently showed that basophils, in an IgE and IL-4 dependent manner, contribute to SLE pathogenesis by amplifying autoantibody production. Here, we summarize what we have learned about the deleterious role of basophils in lupus both in a mouse model and in SLE patients. We discuss which possible pathways could be involved in basophil activation and recruitment to secondary lymphoid organs during SLE, and how basophils may amplify autoantibody production.
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