Dissection of CrRLK1L signalling pathways regulating disease susceptibility and resistance to powdery mildew (B09)

本项目研究拟南芥CrRLK1L FERONIA及其RALF肽配体支持白粉病感染的分子机制。CrRLK1Ls通过一个基因家族内的易感和抗性因子影响白粉病的侵袭。本项目将研究拟南芥CrRLK1L和RALF多样性在白粉病侵袭过程中的差异调控功能及其与FERONIA的遗传生化相互作用。

This project examines the molecular mechanisms of the Arabidopsis CrRLK1L FERONIA, together with its RALF peptide ligands, supporting powdery mildew infection. Additional CrRLK1Ls affect powdery mildew invasion with susceptibility and resistance factors within one gene family. This project will study Arabidopsis CrRLK1L and RALF diversity underlying differential regulatory functions during powdery mildew invasion and their genetic and biochemical interplay with FERONIA.