Molecular analysis of the relation between R-protein and morphogenesis in plant

组成型活性R蛋白UNI-1D诱导拟南芥芽形态发生异常。我们已经分离出UNI-1D的抑制子。其中一个编码RPT2，它是26S蛋白酶体的一个组成部分，直接与UNI结合。直立体是质膜中的一种受体激酶，也是抑制因子之一。然而，在SAM外表达的ER家族基因会影响SAM的活性。韧皮部中表达的ER影响花序茎的伸长，而在内胚层细胞中表达的EPFL4和6在这一过程中起着配基的作用。

The constitutive active R-protein, uni-1D, induced abnormal morphogenesis in Arabidopsis shoot. We have isolated suppressors of uni-1D. One of them encodes RPT2, which is a component of 26S proteasome and binds directly to UNI. Erecta, which is a receptor kinase in a plasma membrane, is also one of the suppressors. ER family genes expressed outside of SAM, however, affect the SAM activity. ER expressed in phloem affect the inflorescence stem elongation and the EPFL4 and 6, both of which expressed in endoderm cells, function as a ligand in this process.