FOOD SELECTION AND REGULATION IN EATING DISORDERS

启动、维持和终止进食的过程 无序的个人还没有被确认。我们建议 评估神经性厌食症、暴食症受试者的反应 神经症，或慢性节食行为对能量的操纵 密度、食物类型、份量、剥夺和食物选择 在住院前后的自然和实验室环境中 治疗。将检验四个主要假设：第一， 进食障碍者的进食行为 在数量上与正常人群的不同 摄取率、饮食选择和发病时间等参数 饱足的感觉。其次，饮食紊乱的受试者未能 根据能量密度调整摄入量 无序的受试者。第三，这些群体将表现出差异性 特定于感觉的饱腹感的发展和对 试餐后对食物的情感和认知变量 消费。最后，记录了异常特征 饮食紊乱的患者将在以下情况下恢复正常 治疗。这些假设将在以下方面得到验证 实验将系统地评估监管、食品 对食物的选择和反应：1.调节热量的能力 摄入量将使用高卡路里和低卡路里预负荷进行调查 或者流质饮食。受试者补偿的程度 卡路里含量将表明对内部监管的敏感性 暗示。2.对熟悉的食物的反应是高或低- 将对卡路里进行研究。食物选择、消耗量、费率 以及对感觉、情感和认知变量的评级 将为基本摄食障碍提供证据 流程。3.三种不同水平的剥夺对儿童心理健康的影响 饮食行为将受到监控。以下进水口的调整 剥夺将表示对内部信号的反应 饥饿。4.自助餐厅的食物选择和能量摄入 实验室设置将在治疗前后进行比较。 恢复正常摄入量水平的程度 都会被记录下来。这些实验将使我们能够识别 表征饮食紊乱的参数。这样的信息 不仅会加深我们对心绞痛发病机制的认识 饮食失调，但将增加我们对其机制的了解 正常人的潜在进食。

The processes which initiate, maintain and terminate eating in disordered individuals have not been identified. We propose to evaluate the responses of subjects with anorexia nervosa, bulimia nervosa, or chronic dieting behavior to manipulations of energy density, food type, portion size, deprivation and food selection in naturalistic and laboratory settings before and after inpatient treatment. Four main hypotheses will be tested: First, that the eating behavior of eating behavior of eating disordered individuals differs quantitatively from that of the normal population on parameters such as rate of ingestion, dietary selection and onset of satiety. Second, that eating disordered subjects fails to adjust intake according to energy density relative to non- disordered subjects. Third, that the groups will show differential development of sensory-specific satiety and changes in rating of affective and cognitive variables to foods after test meal consumption. Finally, that the abnormal characteristics recorded in eating disordered patients will be normalized following treatment. These hypotheses will be tested in the following experiments which will systematically assess regulation, food selection and responses to foods: 1. Ability to regulate caloric intake will be investigated using a high and low-calorie preload or a liquid diet. The degree to which subjects compensate for caloric content will indicate sensitivity to internal regulatory cues. 2. Responses to familiar foods which are high or low- calorie will be studied. Food selection, amount consumed, rate of eating and ratings of sensory, affective and cognitive variables will provide evidence for disturbances of basic ingestive processes. 3. The influence of three levels of deprivation on eating behavior will be monitored. Adjustment of intake following deprivation will indicate responsiveness to internal signals of hunger. 4. Food selection and energy intake in a cafeteria and laboratory setting will be compared before and after treatment. The extent to which normalized levels of intake have been restored will be recorded. These experiments will permit us to identify parameters which characterize disordered eating. Such information will enhance not only our understanding of the pathogenesis of eating disorders but will increase our knowledge of the mechanisms underlying eating in normals.