Associations between traffic noise, particulate air pollution, hypertension, and isolated systolic hypertension in adults: the KORA study.
Associations between traffic noise, particulate air pollution, hypertension, and isolated systolic hypertension in adults: the KORA study.
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DOI：
10.1289/ehp.1306981
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发表时间：
2014-05
影响因子：
10.4
通讯作者：
Peters A
中科院分区：
文献类型：
--
作者：
Babisch W;Wolf K;Petz M;Heinrich J;Cyrys J;Peters A
背景资料：关于交通噪音与心血管疾病之间关系的研究很少将空气污染作为分析中的协变量。单纯收缩期高血压尚未成为流行病学噪声研究的重点。研究方法：在两个研究人群中评估了交通噪音（公路和铁路）与高血压患病率之间的关系，共有4，166名25-74岁的参与者。交通噪音（加权昼夜平均噪音水平; LDN）在住宅的门面来自噪音地图。年平均PM2.5质量浓度在住宅地址估计土地利用回归。通过血压读数、医生诊断的高血压自我报告和降压药物摄入来评估高血压。结果：在大奥格斯堡，德国，研究人群，交通噪音和空气污染与高血压无关。在暴露评估更详细的奥格斯堡市人群（n = 1，893）中，噪声增加10 dB（A）的调整后比值比（OR）为1.16（95% CI：1.00，1.35），在对PM2.5进行额外调整后为1.11（95% CI：0.94，1.30）。PM2.5增加1 μg/m3的调整OR为1.15（95% CI：1.02，1.30），在额外调整噪声后为1.11（95% CI：0.98，1.27）。对于单纯收缩期高血压，噪音和PM2.5的完全校正OR分别为1.43（95% CI：1.10，1.86）和1.08（95% CI：0.87，1.34）。结论：交通噪声和PM2.5均与高血压患病率相关。与高血压的相互校正关联是积极的，但不再具有统计学意义。引文：Babisch W，Wolf K，Petz M，Heinrich J，Cyrys J，Peters A. 2014.交通噪声、空气颗粒污染、高血压和成人单纯收缩期高血压之间的关系：KORA研究环境健康展望122：492-498; http：dx.doi.org/10.1289/ehp.1306981
Background: Studies on the association between traffic noise and cardiovascular diseases have rarely considered air pollution as a covariate in the analyses. Isolated systolic hypertension has not yet been in the focus of epidemiological noise research. Methods: The association between traffic noise (road and rail) and the prevalence of hypertension was assessed in two study populations with a total of 4,166 participants 25–74 years of age. Traffic noise (weighted day–night average noise level; LDN) at the facade of the dwellings was derived from noise maps. Annual average PM2.5 mass concentrations at residential addresses were estimated by land-use regression. Hypertension was assessed by blood pressure readings, self-reported doctor-diagnosed hypertension, and antihypertensive drug intake. Results: In the Greater Augsburg, Germany, study population, traffic noise and air pollution were not associated with hypertension. In the City of Augsburg population (n = 1,893), where the exposure assessment was more detailed, the adjusted odds ratio (OR) for a 10-dB(A) increase in noise was 1.16 (95% CI: 1.00, 1.35), and 1.11 (95% CI: 0.94, 1.30) after additional adjustment for PM2.5. The adjusted OR for a 1-μg/m3 increase in PM2.5 was 1.15 (95% CI: 1.02, 1.30), and 1.11 (95% CI: 0.98, 1.27) after additional adjustment for noise. For isolated systolic hypertension, the fully adjusted OR for noise was 1.43 (95% CI: 1.10, 1.86) and for PM2.5 was 1.08 (95% CI: 0.87, 1.34). Conclusions: Traffic noise and PM2.5 were both associated with a higher prevalence of hypertension. Mutually adjusted associations with hypertension were positive but no longer statistically significant. Citation: Babisch W, Wolf K, Petz M, Heinrich J, Cyrys J, Peters A. 2014. Associations between traffic noise, particulate air pollution, hypertension, and isolated systolic hypertension in adults: the KORA Study. Environ Health Perspect 122:492–498; http://dx.doi.org/10.1289/ehp.1306981
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