Can Chronic Remote Cortical Hypoperfusion Induced by Thalamic Infarction Cause Damage of Tracts Passing through Those Hypoperfused Regions?
Can Chronic Remote Cortical Hypoperfusion Induced by Thalamic Infarction Cause Damage of Tracts Passing through Those Hypoperfused Regions?
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DOI：
10.3389/fneur.2013.00156
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发表时间：
2013
影响因子：
3.4
通讯作者：
Berger E
中科院分区：
文献类型：
--
作者：
Magnin E;Chamard L;Vuillier F;Tatu L;Berger E
我们报告的情况下，一名妇女提出的变化，脑成像后一年半的双丘脑（主要是左侧）梗死，包括外侧和内侧丘脑核。外侧膝状体和枕无损伤。在左侧皮质和基底结构中观察到低灌注。卒中后1年半，左侧半球和枕部出现白色高信号病变。丘脑内侧核和外侧核与枕叶皮质的联系并不紧密。因此，除了丘脑卒中后的沃勒变性，我们假设由神经联系不能引起的慢性左颞叶低灌注可导致通过颞叶的枕叶纤维束（视辐射）的单侧慢性缺氧损伤。
We report the case of a woman presenting with changes on cerebral imaging a year and a half after a bi-thalamic (predominantly left-sided) infarction including lateral and medial thalamic nuclei. Lateral geniculate body and pulvinar were not damaged. Hypoperfusion was observed in left cortical and basal structures. White matter FLAIR hyperintense lesions occurred in the left hemisphere and the occipital region 1 year and half after stroke. Medial and lateral thalamic nuclei are not highly connected to the occipital cortex. Therefore, in addition to Wallerian degeneration after thalamic stroke, we hypothesize that the chronic left temporal hypoperfusion induced by diaschisis can lead to a lateralized chronic hypoxic damage of the occipital fiber tract (optic radiation) that passes through the temporal lobe.
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影响因子：
3.7
作者：
Johansen-Berg, H;Behrens, TEJ;Matthews, PM
通讯作者：
Matthews, PM
影响因子：
2.9
作者：
Chida, Kohei;Ogasawara, Kuniaki;Ogawa, Akira
通讯作者：
Ogawa, Akira
影响因子：
2.9
作者：
De Reuck, J;Stevens, H;Korf, J
通讯作者：
Korf, J
影响因子：
2.9
作者：
Iglesias, S;Marchal, G;Baron, JC
通讯作者：
Baron, JC
影响因子：
3.5
作者：
Stenset, V.;Grambaite, R.;Fladby, T.
通讯作者：
Fladby, T.