Slow-Speed Low-Intensity but Not Normal-Speed High-Intensity Resistance Exercise Maintains Endothelial Function
Slow-Speed Low-Intensity but Not Normal-Speed High-Intensity Resistance Exercise Maintains Endothelial Function
复制标题
慢速低强度而非正常速度高强度阻力运动可维持内皮功能
DOI:
10.1080/02701367.2021.2022586
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发表时间:
2022
影响因子:
2.2
通讯作者:
Eisuke Ochi
中科院分区:
文献类型:
--
作者:
Yosuke Tsuchiya;Takuma Morishima;Eisuke Ochi
Purpose: High-intensity resistance exercise two or three times a week has been considered optimal for muscle hypertrophy, although it can remarkably elevate blood pressure (BP). In contrast, slow-speed resistance exercise with low intensity and tonic force generation (slow-low) can induce muscle hypertrophy without elevating BP. However, it is unclear how endothelial function changes after slow-low. Therefore, this study examined whether slow-low would maintain brachial artery endothelial function in comparison with normal-speed with high intensity resistance exercise (normal-high) and normal-speed with low-intensity resistance exercise (normal-low).Methods: Eleven healthy young men performed leg-extensions with slow-low (3 sets of 8 repetitions at 50% of 1RM), normal-high (3 sets of 8 repetitions at 80% of 1RM), and normal-low (3 sets of 8 repetitions at 50% of 1RM). Flow-mediated dilation (FMD) in the brachial artery was evaluated at pre-exercise and at 10, 30, and 60 min after exercise.Result: The results showed that normal-high caused significant impairment of FMD at 30 (3.7 ± 2.7%) and 60 (3.7 ± 2.8%) min after exercise (P < .05). In contrast, slow-low and normal-low showed no significant difference from baseline. FMD was significantly lower in normal-high compared with slow-low and normal-low at 30 and 60 min after exercise (P < .05). Additionally, systolic BP was significantly higher during normal-high compared with slow-low and normal-low (P < .05).Conclusion: We concluded that slow-low did not impair brachial artery FMD concomitant with lower systolic BP, and may therefore be a useful mode of exercise training to improve muscle hypertrophy without provoking transient endothelial dysfunction.
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DOI:
10.1126/sageke.2004.6.nf18
发表时间:
2004
期刊:
Science of aging knowledge environment : SAGE KE
影响因子:
--
作者:
M. Leslie
通讯作者:
M. Leslie
DOI:
10.1249/01.mss.0000175865.17614.74
发表时间:
2005-11-01
期刊:
MEDICINE AND SCIENCE IN SPORTS AND EXERCISE
影响因子:
--
作者:
Jurca, R;Lamonte, MJ;Blair, SN
通讯作者:
Blair, SN
影响因子:
3.3
作者:
MACDOUGALL, JD;MCKELVIE, RS;BUICK, F
通讯作者:
BUICK, F
影响因子:
18.4
作者:
Steven T. Linsenbardt;T. R. Thomas;Richard W. Madsen PhDt
通讯作者:
Richard W. Madsen PhDt
影响因子:
3.3
作者:
Takarada, Y;Nakamura, Y;Ishii, N
通讯作者:
Ishii, N