JPP Student Journal Club Commentary: Linking Biology to the Environment: Novel Methods for Understanding Pediatric Obesity.
JPP Student Journal Club Commentary: Linking Biology to the Environment: Novel Methods for Understanding Pediatric Obesity.
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JPP 学生期刊俱乐部评论:将生物学与环境联系起来:了解儿童肥胖的新方法。
DOI:
10.1093/jpepsy/jsy001
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发表时间:
2018
影响因子:
3.6
通讯作者:
Black,MaureenM
中科院分区:
文献类型:
--
作者:
Armstrong,Bridget;Black,MaureenM
Pediatric obesity is a complex, seemingly intransient public health concern that has spurred research to identify causes and potential solutions. Both obesityrelated behaviors and outcomes are affected by dynamic processes of many factors across multiple levels of the social ecological model. The environmental–behavioral relationship is reciprocal, dynamic, and dominated by temporally dependent feedback. Pediatric psychology and public health have generated substantial literature on predictors of obesity, with pediatric psychology focusing on individual social determinates and public health focusing on environmental determinants. Only limited research has integrated environment and individual predictors, despite calls for integration (Black & Hager, 2013; McGuire, 2012). The study by Gartstein and colleagues (Gartstein, Seamon, Thompson, & Lengua, 2018), published in this issue, successfully integrated individual biological data and environmental context. Comprehending and mitigating the complexity of obesity will likely require a combination of environmental and individual data. Advances in technology have enhanced the ability to gather objective individual-level data. Examples in pediatric psychology alone include accelerometers (Cushing et al., 2017), ecological momentary assessment (Heron, Everhart, McHale, & Smyth, 2017), magnetic resonance imaging (Jensen, Duraccio, Carbine, & Kirwan, 2016), continuous glucose monitoring (Nelson, Aylward, & Rausch, 2011), and cortisol sampling (McCarthy et al., 2011). Technology has also improved the collection and sharing of spatial environmental data. Spatial data include governmentfunded efforts (ie, census data), private data (ie, business mailing lists from InfoUSA), and publicly available proprietary data (ie, Google street view; Odgers, Caspi, Bates, Sampson, & Moffitt, 2012). Spatial data are often publicly available through data repositories (ie, https://www. communitycommons. org/), enabling the integration of individual indicators of health (ie, individual cortisol rates) with “community context,” such as the interaction between cortisol and crime rates used by Gartstein et al.(2018).A cautionary note is that data quality varies across sources. For example, crime data include errors of underreporting (not all crimes are reported) and misclassification (not all crimes are reported accurately, or at all)(Nolan, Haas, & Napier, 2011). Under ideal conditions, high-quality spatial data would be longitudinal, include descriptions of how and when data were collected, and be fine-grain enough to analyze small geographic areas. Examining environmental changes over time can elucidate the bidirectional effects of individual and environmental determinants of health. Because the spatial data used by Gartstein et al.(2018) were restricted to the Seattle area for 2008, the authors could not use the longitudinal cortisol data that they had collected. The study would have been strengthened by the inclusion of more spatial and temporal information to match their longitudinal individual-level data. Future increases in the amount and quality of publicly available time-specific, longitudinal spatial data will enable the integration of rich individual data from pediatric psychology studies with environmental data.
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DOI:
10.1111/j.1469-7610.2012.02565.x
发表时间:
2012-10
期刊:
Journal of child psychology and psychiatry, and allied disciplines
影响因子:
--
作者:
Odgers CL;Caspi A;Bates CJ;Sampson RJ;Moffitt TE
通讯作者:
Moffitt TE
DOI:
10.1056/nejmsa1603542
发表时间:
2017-05-11
期刊:
The New England journal of medicine
影响因子:
--
作者:
Coady SA;Mensah GA;Wagner EL;Goldfarb ME;Hitchcock DM;Giffen CA
通讯作者:
Giffen CA
影响因子:
3.6
作者:
Palermo,TonyaM;Janicke,DavidM;McQuaid,ElizabethL;Mullins,LarryL;Robins,PaulM;Wu,YelenaP
通讯作者:
Wu,YelenaP
影响因子:
3.6
作者:
Cushing, Christopher C.;Mitchell, Tarrah B.;Noser, Amy E.
通讯作者:
Noser, Amy E.
影响因子:
3.6
作者:
J. Nolan;S. Haas;Jessica S. Napier
通讯作者:
Jessica S. Napier