Endoscopic Features of Gastric Epithelial Neoplasm of Fundic Gland Mucosa Lineage.
Endoscopic Features of Gastric Epithelial Neoplasm of Fundic Gland Mucosa Lineage.
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DOI:
10.3390/diagnostics12112666
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发表时间:
2022-11-02
期刊:
影响因子:
3.6
通讯作者:
Nagahara, Akihito
中科院分区:
文献类型:
--
作者:
Matsumoto, Kohei;Ueyama, Hiroya;Yao, Takashi;Iwano, Tomoyo;Yamamoto, Momoko;Utsunomiya, Hisanori;Uchida, Ryota;Abe, Daiki;Oki, Shotaro;Suzuki, Nobuyuki;Ikeda, Atsushi;Yatagai, Noboru;Akazawa, Yoichi;Takeda, Tsutomu;Ueda, Kumiko;Asaoka, Daisuke;Hojo, Mariko;Nagahara, Akihito
关键词:
The endoscopic features of gastric epithelial neoplasms of fundic gland mucosa lineage (GEN-FGML) have not been well investigated. We aimed to clarify the endoscopic features of GEN-FGML and differences between gastric adenocarcinoma of the fundic gland type (GA-FG) and fundic gland mucosa type (GA-FGM). A total of 62 GEN-FGML lesions, including 52 GA-FG and 10 GA-FGM, were retrospectively analyzed using endoscopic and clinicopathological findings to provide information of diagnostic value using white light imaging (WLI) and magnifying endoscopy with narrow-band imaging (M-NBI). GA-FG frequently presented with a whitish, submucosal tumor (SMT) shape with dilated vessels with branching architecture and background mucosa without atrophic change in WLI, an indistinct demarcation line (DL), dilatation of the crypt opening and intervening part (IP), and microvessels without distinct irregularity in M-NBI. GA-FGM frequently presented as a reddish, elevated lesion in WLI, with a distinct DL, dilatation of the IP, and an irregular microvascular pattern in M-NBI. As for an M-NBI diagnosis, five GA-FGM lesions met the diagnostic criteria for cancer, whereas none of the GA-FG lesions met the same criteria. We highlight the endoscopic features of GEN-FGML, and the differentiation between GA-FG and GA-FGM might be possible by combination of lesion color and morphology in WLI and M-NBI diagnoses.
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影响因子:
6.3
作者:
Ueyama H;Yao T;Akazawa Y;Hayashi T;Kurahara K;Oshiro Y;Yamada M;Oda I;Fujioka S;Kusumoto C;Fukuda M;Uchita K;Kadota T;Oono Y;Okamoto K;Murakami K;Matsuo Y;Kato M;Maehata T;Yahagi N;Yasuhara Y;Yada T;Uraushihara K;Yamane T;Matsuo T;Ito M;Maruyama Y;Osako A;Ono S;Kato M;Yagi K;Hashimoto T;Tomita N;Tsuyama S;Saito T;Matsumoto K;Matsumoto K;Watanabe S;Uemura N;Chiba T;Nagahara A
通讯作者:
Nagahara A
影响因子:
1.6
作者:
Takahashi K;Fujiya M;Ichihara S;Moriichi K;Okumura T
通讯作者:
Okumura T
影响因子:
5.6
作者:
Ueyama, Hiroya;Yao, Takashi;Watanabe, Sumio
通讯作者:
Watanabe, Sumio
影响因子:
2.6
作者:
Matsumoto K;Ueyama H;Yao T;Abe D;Oki S;Suzuki N;Ikeda A;Yatagai N;Akazawa Y;Komori H;Takeda T;Matsumoto K;Hojo M;Nagahara A
通讯作者:
Nagahara A
DOI:
10.3760/cma.j.cn112151-20190720-00404
发表时间:
2020-04-08
期刊:
Zhonghua bing li xue za zhi = Chinese journal of pathology
影响因子:
--
作者:
Sun, W W;Zhang, L;Da, Q
通讯作者:
Da, Q