Marital quality and health: a meta-analytic review.
Marital quality and health: a meta-analytic review.
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DOI：
10.1037/a0031859
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发表时间：
2014-01
影响因子：
22.4
通讯作者：
McGinn, Meghan M.
中科院分区：
文献类型：
--
作者：
Robles, Theodore F.;Slatcher, Richard B.;Trombello, Joseph M.;McGinn, Meghan M.
这项元分析回顾了过去50年发表的126篇经验性文章，这些文章描述了超过72,000人的婚姻关系质量和身体健康之间的关系。健康结果包括临床终点(对功能、疾病严重性和死亡率的客观评估；主观健康评估)和替代终点(替代临床终点的生物标记物，如血压)。生物介质包括心血管反应性和下丘脑-垂体-肾上腺轴活性。更高的婚姻质量与更好的健康相关，平均效应大小在r=0.07到0.21之间，包括较低的死亡风险，r=.11，以及婚姻冲突期间较低的心血管反应性，r=−.13，但与冲突期间的每日皮质醇斜率或皮质醇反应性无关。小效应的大小与先前发现的健康行为(例如，饮食)和健康结果之间的关联在大小上相似。临床结果的一小部分的效应大小容易受到发表偏倚的影响。在一些研究中，在考虑了年龄和社会经济地位等混杂因素后，效应大小仍然显著。样本中女性比例较高的研究显示了更大的效应大小，但我们在明确测试性别适度的研究中几乎没有发现性别差异的证据，替代终点研究除外。我们的结论受到针对特定健康结果的少量研究、无法解释的异质性以及限制因果推断的设计的限制。这些发现强调了在未来的研究中明确测试情感、健康行为和生物机制的必要性，并将重点放在可能改变婚姻质量和健康之间关系的调节因素上。
This meta-analysis reviewed 126 published empirical articles over the past 50 years describing associations between marital relationship quality and physical health in over 72,000 individuals. Health outcomes included clinical endpoints (objective assessments of function, disease severity, and mortality; subjective health assessments) and surrogate endpoints (biological markers that substitute for clinical endpoints, such as blood pressure). Biological mediators included cardiovascular reactivity and hypothalamic-pituitary-adrenal axis activity. Greater marital quality was related to better health, with mean effect sizes from r = .07 to .21, including lower risk of mortality, r = .11, and lower cardiovascular reactivity during marital conflict, r = −.13, but not daily cortisol slopes or cortisol reactivity during conflict. The small effect sizes were similar in magnitude to previously found associations between health behaviors (e.g., diet) and health outcomes. Effect sizes for a small subset of clinical outcomes were susceptible to publication bias. In some studies, effect sizes remained significant after accounting for confounds such as age and socioeconomic status. Studies with a higher proportion of women in the sample demonstrated larger effect sizes, but we found little evidence for gender differences in studies that explicitly tested gender moderation, with the exception of surrogate endpoint studies. Our conclusions are limited by small numbers of studies for specific health outcomes, unexplained heterogeneity, and designs that limit causal inferences. These findings highlight the need to explicitly test affective, health behavior, and biological mechanisms in future research, and focus on moderating factors that may alter the relationship between marital quality and health.
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影响因子：
4.7
作者：
Appelberg, K;Romanov, K;Koskenvuo, M
通讯作者：
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影响因子：
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作者：
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