TPPU protects tau from H2O2-induced hyperphosphorylation in HEK293/tau cells by regulating PI3K/AKT/GSK-3β pathway
TPPU protects tau from H2O2-induced hyperphosphorylation in HEK293/tau cells by regulating PI3K/AKT/GSK-3β pathway
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TPPU 通过调节 PI3K/AKT/GSK-3β 途径保护 HEK293/tau 细胞中 tau 免受 H<sub>2</sub>O<sub>2</sub> 诱导的过度磷酸化
DOI:
10.1007/s11596-016-1662-z
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发表时间:
2016
期刊:
影响因子:
--
通讯作者:
Wang MH
中科院分区:
文献类型:
--
作者:
Yao ES;Tang Y;Liu XH;Wang MH
Neurofibrillary pathology of abnormally hyperphosphorylated tau is a hallmark of Alzheimer’s disease (AD) and other tauopathies. Phosphatidylinositol 3-kinase (PI3K)/Akt/glycogen synthase kinase-3 beta (GSK-3β) signaling pathway is pivotal for tau phospho
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