Trimeric autotransporter adhesins contribute to Actinobacillus pleuropneumoniae pathogenicity in mice and regulate bacterial gene expression during interactions between bacteria and porcine primary alveolar macrophages

Trimeric autotransporter adhesins contribute to Actinobacillus pleuropneumoniae pathogenicity in mice and regulate bacterial gene expression during interactions between bacteria and porcine primary alveolar macrophages
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三聚体自转运蛋白粘附素有助于小鼠胸膜肺炎放线杆菌的致病性,并在细菌与猪原代肺泡巨噬细胞相互作用期间调节细菌基因表达

DOI:
10.1007/s10482-015-0609-x
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发表时间:
2015-10
期刊:
Antonie van Leeuwenhoek
影响因子:
--
通讯作者:
Lei Liancheng
Lei Liancheng
中科院分区:
其他
文献类型:
--
作者:
Qin Wanhai;Lei Liancheng

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胸膜肺炎放线杆菌是引起猪呼吸道疾病的重要病原菌。三聚体自身转运蛋白粘附素(TAA)是新近发现的一种介导细菌黏附和定植的细菌毒力因子。两个TAA编码
Actinobacillus pleuropneumoniae is an important pathogen that causes respiratory disease in pigs. Trimeric autotransporter adhesin (TAA) is a recently discovered bacterial virulence factor that mediates bacterial adhesion and colonization. Two TAA coding
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