Increased insomnia symptoms predict the onset of back pain among employed adults.

Increased insomnia symptoms predict the onset of back pain among employed adults.
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DOI:
10.1371/journal.pone.0103591
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发表时间:
2014
期刊:
影响因子:
3.7
通讯作者:
Armon G
Armon G
中科院分区:
综合性期刊3区
文献类型:
--
作者:
Agmon M;Armon G

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背痛是导致成人慢性残疾的最普遍的疼痛疾病之一,失眠是常见的合并症。然而,失眠是否先于背痛或反之亦然仍然不清楚。目前的研究测试了失眠和背痛之间的时间关联。采用纵向设计研究失眠随时间的变化是否能预测背痛的发作,反之亦然。该研究在3个时间点(T1、T2和T3)对一组活跃的健康工作成年人(N = 2,131,34%为女性)进行,为期3.7年(范围= 2.2 - 5.12)。    Logistic回归分析用于测试从T1到T2的失眠症状增加是否预示着新背痛的发生。普通最小二乘回归用于测试T2时背痛的存在是否预示着从T2到T3失眠的增加。结果表明,在控制了社会经济变量、自我报告的健康状况、生活方式行为和人体测量学后,T1-T2失眠症状的增加与T3背痛风险增加1.40倍相关(OR = 1.40; 95% CI = 1.10 - 1.71)。    没有发现反向因果关系的支持;即,背痛预示着失眠的增加。肌肉紧张似乎是健康个体发生背痛的一个危险因素。然而,没有发现反向因果关系的证据。
Back pain is among the most prevalent pain disorders causing chronic disability among adults, and insomnia is a common co-morbidity. However, whether insomnia precedes back pain or vice versa remains unclear. The current study tested the temporal association between insomnia and back pain. A longitudinal design was used to investigate whether changes in insomnia over time predict the onset of back pain and vice versa. The study was conducted on a cohort of active healthy working adults (N = 2,131, 34% women) at three time points (T1, T2, and T3) over a period of 3.7 years (range = 2.2–5.12) years. Logistic regression analysis was used to test whether increased insomnia symptoms from T1 to T2 predicted the onset of new back pain. Ordinary least squares regression was used to test whether the existence of back pain at T2 predicted an increase in insomnia from T2 to T3. The results indicated that after controlling for socioeconomic variables, self-reported health, lifestyle behaviors, and anthropometrics, a T1–T2 increase in insomnia symptoms was associated with a 1.40-fold increased risk of back pain at T3 (OR = 1.40; 95% CI = 1.10–1.71). No support was found for reverse causation; i.e., that back pain predicts subsequent increase in insomnia. Insomnia appears to be a risk factor in the development of back pain in healthy individuals. However, no evidence of reverse causation was found.
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