Identifying muscle contraction evoked mechanisms that counteract diet-induced insulin resistance

识别肌肉收缩诱发机制，抵消饮食引起的胰岛素抵抗

• 批准号: 202890
• 负责人: Kewalramani Girish R
• 金额: $ 9.83万
• 依托单位: Hospital for Sick Children (Toronto)
• 依托单位国家: 加拿大
• 项目类别: Fellowship Programs
• 财政年份: 2010
• 资助国家: 加拿大
• 起止时间: 2010-02-01 至 2013-02-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=202890&lang=en
• 关键词: Contraction; Diabetes; Immunology; Inflammation; Insulin Resistance; Macrophages; Obesity; Skeletal Muscle

Insulin controls blood sugar and is required for the entry of sugar (glucose) into blood cells from blood. Excess body fat in obesity or due to high fat diet makes body resistant to insulin. Due to this insulin resistance, sugar accumulates in the blood a

胰岛素控制血糖，是糖（葡萄糖）从血液进入血细胞所必需的。肥胖或高脂肪饮食导致的体内脂肪过多会使身体对胰岛素产生抵抗。由于这种胰岛素抵抗，糖在血液中积累，