TLR3 is an endogenous sensor of cell death and a potential target for induction of long-term heart transplant survival

TLR3 是细胞死亡的内源性传感器，也是诱导心脏移植长期存活的潜在靶标

• 批准号: 409932
• 负责人: Zhang Zhu-Xu
• 金额: $ 40.13万
• 依托单位: University of Western Ontario
• 依托单位国家: 加拿大
• 项目类别: Operating Grants
• 财政年份: 2019
• 资助国家: 加拿大
• 起止时间: 2019-09-01 至 2024-09-01
• 项目状态: 已结题
• 来源: https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=409932&lang=en
• 关键词: Apoptosis; Injury; Necroptosis; Transplantation

When a heart is irreversibly damaged and cannot be treated by any other medical or surgical means, organ transplantation is the most beneficial and highly-pursued treatment option for saving the person's life. However, the immune system of the graft recip

当心脏受到不可逆的损伤，无法通过任何其他医疗或手术手段治疗时，器官移植是挽救生命的最有益和最受欢迎的治疗选择。然而，移植物受体的免疫系统