Relaxin: molecular mechanisms of action in the reversal of fibrosis

松弛素：逆转纤维化的分子作用机制

• 批准号: LP0560620
• 负责人: Prof Roger Summers
• 金额: $ 26.58万
• 依托单位: Monash University
• 依托单位国家: 澳大利亚
• 项目类别: Linkage Projects
• 财政年份: 2005
• 资助国家: 澳大利亚
• 起止时间: 2005-10-07 至 2007-12-31
• 项目状态: 已结题
• 来源: https://dataportal.arc.gov.au/RGS/Web/Grants/LP0560620
• 关键词: Relaxin; molecular; mechanisms; action; reversal

Defects in relaxin and relaxin receptors are increasingly implicated as a cause of fibrosis which is associated with many disease processes. This study will examine the molecular mechanisms linking relaxin and fibrosis and will determine whether relaxin can be used to reverse the condition.

松弛素及其受体的缺陷越来越多地被认为是导致纤维化的一个原因，而纤维化与许多疾病过程有关。这项研究将研究松弛素和纤维化之间的分子机制，并将确定松弛素是否可以用来逆转这种情况。