Mathematical Modeling of the Marxian Economics Based on Neoclassical Growth Model
基于新古典增长模型的马克思经济学数学模型
基本信息
- 批准号:16530115
- 负责人:
- 金额:$ 1.92万
- 依托单位:
- 依托单位国家:日本
- 项目类别:Grant-in-Aid for Scientific Research (C)
- 财政年份:2004
- 资助国家:日本
- 起止时间:2004 至 2006
- 项目状态:已结题
- 来源:
- 关键词:
项目摘要
In this research, we extended our Marxian Optimal Growth Theory which was first established our paper in 2002 into various fields.The first field is improvements of our model for the two-class model although our original model was one-class model, by these improvements now we are able to study the dynamics of exploitation in the growth path. Especially, our main achievement is that class division makes over-accumulation under certain conditions, and to avoid it, society tend to cause social democrats, anti-productivism and trade unionism as well as Marxism. We also found some conditions which diminish exploitation under zero-growth society and its relationship with labor theory of value.The second field is the extensions of the one-class model itself. For example, change of the production function of the productive goods to have capital goods as its means of production, extension from the social planner model to decentralized market-oriented model, extension from the Industrial revolution model to the Agricultural revolution model or Knowledge revolution model, extension from the deterministic model to the stochastic model, extension to explain the present various phenomena of zero growth society in Japan.The third field is the study of the relations of our definition of exploitation with Roemer's, and what conditions make the former same as the latter. This study opened new field.
在本研究中,我们将2002年提出的马克思最优增长理论扩展到各个领域:第一个领域是对两类模型的改进,虽然我们原来的模型是一类模型,但通过这些改进,我们现在能够研究增长路径中的剥削动态,特别是,我们的主要成果是,阶级分化在一定条件下造成过度积累,为了避免过度积累,社会倾向于产生社会民主主义、反生产主义和工会主义以及马克思主义。我们还发现了零增长社会中减少剥削的条件及其与劳动价值论的关系。第二个领域是一级模型本身的扩展。例如,将生产资料的生产函数转变为以资本资料作为生产资料,从社会计划模型扩展到分散的市场导向模型,从工业革命模型扩展到农业革命模型或知识革命模型,从确定性模型扩展到随机模型,第三个领域是研究我们的剥削定义与罗默的剥削定义之间的关系,以及在什么条件下使前者与后者相同。该研究开辟了新的领域。
项目成果
期刊论文数量(48)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)
関干馬克思最優増長論的解釈-最優迂回生産程序的資本主義数学模型
关干马克志对最优增长理论的诠释——基于最优生产过程的资本主义数学模型
- DOI:
- 发表时间:2005
- 期刊:
- 影响因子:0
- 作者:山下裕歩;大西広;茹仙古麗;吾甫尓
- 通讯作者:吾甫尓
規範理論としての労働搾取論-吉原直毅氏による「マルクスの基本定理」批判再論
作为规范理论的劳动剥削理论——吉原直树对“马克思基本定理”的批判性重新审视
- DOI:
- 发表时间:2007
- 期刊:
- 影响因子:0
- 作者:藪下史郎;荒木一法;石川 健;松尾匡
- 通讯作者:松尾匡
Hiroshi Ohnishi and Roxiangul Uful, Reconstruction Marxism as a Neoclassical Optimal Growth Model
Hiroshi Ohnishi 和 Royangul Uful,将马克思主义重建为新古典最优增长模型
- DOI:
- 发表时间:2005
- 期刊:
- 影响因子:0
- 作者:大西広;山下裕歩;茹仙古麗吾甫尓;Hiroshi Ohnishi;Hiroshi Ohnishi;Yuuho Yamashita
- 通讯作者:Yuuho Yamashita
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OHNISHI Hiroshi其他文献
Accumulation of dysregulated renal mononuclear phagocytes (rMoPh) and Th1 cells in the kidney of CD11c-specific Shp-1 knockout mice
CD11c 特异性 Shp-1 敲除小鼠肾脏中失调的肾单核吞噬细胞 (rMoPh) 和 Th1 细胞的积累
- DOI:
- 发表时间:
2016 - 期刊:
- 影响因子:0
- 作者:
WATANABE Mitsuharu;KANEKO Yoriaki;HIROMURA Keiju;KINOSHITA Masato;OHISHI Yuko;SAITO Yasuyuki;OHNISHI Hiroshi;MATOZAKI Takashi;NOJIMA Yoshihisa - 通讯作者:
NOJIMA Yoshihisa
Dendritic cell-specific ablation of the protein tyrosine phosphatase Shp-1 induces autoimmune sialadenitis characterized by infiltration of CD4+ T cells and B cells
树突状细胞特异性去除蛋白酪氨酸磷酸酶Shp-1可诱导自身免疫性唾液腺炎,其特征是CD4 T细胞和B细胞浸润
- DOI:
- 发表时间:
2017 - 期刊:
- 影响因子:0
- 作者:
KINOSHITA Masato;KANEKO Yoriaki;WATANABE Mitsuharu,OHISHI Yuko;SAITO Yasuyuki;OHNISHI Hiroshi;NOJIMA Yoshihisa;MATOZAKI Takashi;HIROMURA Keiju - 通讯作者:
HIROMURA Keiju
Shp-1 in dendritic cells controls the development of memory-phenotype CD8+ T cells
树突状细胞中的 Shp-1 控制记忆表型 CD8 T 细胞的发育
- DOI:
- 发表时间:
2016 - 期刊:
- 影响因子:0
- 作者:
OHISHI Yuko;KANEKO Yoriaki;WATANABE Mitsuharu;KINOSHITA Masato;HIROMURA Keiju;SAITO Yasuyuki;OHNISHI Hiroshi;MATOZAKI Takashi;NOJIMA Yoshihisa - 通讯作者:
NOJIMA Yoshihisa
Dendritic cell-specific ablation of the protein tyrosine phosphatase Shp1 induces enhanced production of inflammatory cytokines by toll-like receptor-mediated stimulation
树突状细胞特异性去除蛋白质酪氨酸磷酸酶Shp1,通过Toll样受体介导的刺激,诱导炎症细胞因子的产生增强
- DOI:
- 发表时间:
2015 - 期刊:
- 影响因子:0
- 作者:
OHISHI Yuko;KANEKO Yoriaki;WATANABE Mitsuharu;KINOSHITA Masato;HIROMURA Keiju;SAITO Yasuyuki;OHNISHI Hiroshi;MATOZAKI Takashi;NOJIMA Yoshihisa - 通讯作者:
NOJIMA Yoshihisa
OHNISHI Hiroshi的其他文献
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{{ truncateString('OHNISHI Hiroshi', 18)}}的其他基金
Analysis of the signaling mechanisms that regulates brain stress responses through protein tyrosine phosphorylation
通过蛋白质酪氨酸磷酸化调节脑应激反应的信号机制分析
- 批准号:
23500437 - 财政年份:2011
- 资助金额:
$ 1.92万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
The role of visceral adipose tissue CD8+ T cells in obese murine asthma model
内脏脂肪组织CD8 T细胞在肥胖小鼠哮喘模型中的作用
- 批准号:
23591120 - 财政年份:2011
- 资助金额:
$ 1.92万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
Analysis of the role of a protein tyrosine phosphatase in regulation of neuronal functions
蛋白酪氨酸磷酸酶在神经元功能调节中的作用分析
- 批准号:
20500332 - 财政年份:2008
- 资助金额:
$ 1.92万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
Analysis of protein tyrosine phosphorylation signal, which regulates synaptic function.
分析调节突触功能的蛋白质酪氨酸磷酸化信号。
- 批准号:
16500236 - 财政年份:2004
- 资助金额:
$ 1.92万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
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