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Effects of clenching on regulatory function of bite force in man-role of peridontal mechanoreceptor

咬牙对牙周机械感受器人角色咬合力调节功能的影响

基本信息

批准号：
07672134
负责人：
KURASAWA Ikufumi
金额：
$ 1.28万
依托单位：
Matsumoto Dental College
依托单位国家：
日本
项目类别：
Grant-in-Aid for Scientific Research (C)
财政年份：
1995
资助国家：
日本
起止时间：
1995 至 1996
项目状态：
已结题

项目摘要

It has previously been reported that bruxism is noted as one of the important causes of a temporomandibular dysfunction. Various discussions have been done about a relation between muscular activity and bruxism. A negative view is proposed from the result of the resting EMG activity about a causal relationship between the pain and the hyperactivity of the masticatory muscle in patients with tempolomandibular dysfunction. However, the fundamental problem is not clarified how any influence the sutained clenching exerts to the masticatory system. We have investigated whether the clenching that is one of the form of bruxism influence for short term to the stomatognathic system. The conditioning clenching induced a significant error in reproducing a leamed criterion bite force under conditions of no visual feed-back of force, like the experiments of Hutton et al. These findings indicate the possibility that the facilitation of motoneurones of closing muscles might occur after clenching. Furthermore, we have investigated whether the periodontal mechanoreceopors are responsible for the effect after clenching. Following results were obtained. 1. The upper oentral incisors were stimulated by the mechanical stimulator under 5% maximum voluntary teeth clenching. A short-latency inhibitory response was induced in masseter muscle, followed by an exciatory response to brisk taps on the teeth, and mainly excitatory response was induced to slow pushes. 2. These responses were almost abolished by local anesthesia around the teeth stimulated. 3. An excitability response to slow pushes increased with background discharge level. 4. A significant change was not observed for response to slow pushes before and after the clenching for 25% m.v.c.-60 seconds. Consequently, although a reflex arc from the periodontal mechanoreceptor to the masseter motoneurone has relatively little role to the postclenching effect, it is obvious to have a facilitatory effect to slow pushes.

据以往报道，磨牙症是颞下颌关节功能障碍的重要原因之一。关于肌肉活动和磨牙症之间的关系已经进行了各种讨论。从静息肌电活动的结果来看，颞下颌关节功能障碍患者的疼痛与咀嚼肌过度活动之间存在着因果关系。然而，根本的问题是不清楚的任何影响，持续紧握施加到咀嚼系统。本文探讨磨牙症的一种表现形式--紧咬对口颌系统的短期影响。在没有视觉反馈力的条件下，条件性紧咬引起了一个显着的错误，再现leamed标准咬合力，像赫顿等人的实验。这些研究结果表明，关闭肌肉的运动神经元的促进可能发生后，紧咬。此外，我们还调查了牙周机械孔是否对咬牙后的效果负责。获得了以下结果。1.采用机械刺激器刺激上颌中切牙，使其处于5%的最大咬牙强度下。轻叩牙齿引起咬肌短潜伏期抑制反应，轻叩牙齿引起咬肌兴奋反应，轻叩牙齿引起咬肌兴奋反应，轻叩牙齿引起咬肌兴奋反应。2.这些反应几乎被牙齿周围的局部麻醉所消除。3.对缓慢推动的兴奋性反应随着背景放电水平的增加而增加。4.在25% m.v.c.的握紧前后，没有观察到对缓慢推动的反应的显著变化。60秒因此，虽然从牙周机械感受器到咬肌运动神经元的反射弧对后紧效果的作用相对较小，但显然对缓慢推动有促进作用。