Effect of Bite Raising Exerting Neuro-Musclar Mechanism for Musticatory Muscles

在口颌功能障碍患者日益增多的情况下，作为一种广泛应用的保存治疗方法，咬固片的临床意义变得更加重要。然而，一个明确的准则，有关的咬合提高量与bitesprint仍然是不清楚的，但它的行动，甚至提高的生理机制也不清楚。本文首先对咬合力调节机制的动态特性进行了研究，认为咬合力在牙颌功能障碍的发生发展中起着重要作用。实验在6名牙列保存良好的健康成年人上进行。比较两个学习的参考咬合力后紧，不紧。结果，观察到可能与咀嚼肌的紧张、疼痛或其他功能障碍直接相关的持久效果。此外，这是明确的，这一效果的影响，咬rasing的观察是重要的，以探讨bitesprint的作用机制。在未来，我们将考虑涉及的其他问题，与肌肉长度的变化和inpout机制，运动神经元伴随着咬rasing的机械因素。

Clinical implication of bitesprint that is used widely as a preservation therapy, in the present condition that the stomatognathic dysfunction patients are increasing, has become further important. However, a distinct guideline concerning the amount of bite raising with bitesprint remains not only to be unclear, but also even the physiologic mechanism of its action by raising is not clarified.Our research aimed to analyze an influence of bite rasing to bite force. In the begining, we focused on the clenching, being considered to play a causative role in the development of stomatognathic dysfunction, particularly the dynamic properties of regulatory mechanism of bite force after clenching. The experiments were performed on 6 healthy sujects with well-preserved dentitions. Comparison were made between both learned reference bite forces after clenching and no-clenching. As a result, a long lasting effect that might direct relate to tonus, pain or other dysfunctions of the masticatory muscles was observed. Furthermore it was clarified that the observation of bite rasing influence on this effect is important to investigate the mechanism of action of the bitesprint.In future, we will consider the additional problems involved in the mechanical factors associated with muscle length changing and the inpout mechanism to motor neuron accompanied by bite rasing.