Molecular biological Research for exercise prescription with measuring muscle ischemic threshold and Explication for mechanism of exercise induced adaptability.

本研究旨在探讨呼吸、循环系统、骨骼肌等自主分子生物学适应性的推导，以及有氧训练中肌肉收缩力和速度的结合对急性和慢性运动诱导适应性的机制。结果表明，低负荷下的大扭矩运动是控制肺通气和摄氧量的过度条件。此外，在慢性适应方面，高扭矩运动的LT强度及其适应高于低扭矩运动的LT强度和适应速度。在基线和饱和水平下的ltw的差异大于低转矩运动。

The purpose of this study is to examine the derivation of voluntarily molecular biological adaptability such as respiratory and circulatory system, skeletal muscle about mechanism for acute and chronic exercise induced adaptability by combining force and velocity of muscle contraction at aerobic training.Our result showed that high torque exercise at LT work-load rate was the excess condition for controlling pulmonary ventilation and oxygen intake rate as for acute adaptation. In addition, as for chronic adaptation, LT intensity and its adaptation for high torque exercise were higher and faster than those for low torque exercise. The difference of LT watts at baseline and saturated level was higher than that for low torque exercise.