Molecular biological Research for exercise prescription with measuring muscle ischemic threshold and Explication for mechanism of exercise induced adaptability.

本研究的目的是探讨自主分子生物学适应性的来源，如呼吸和循环系统，骨骼肌对急、慢性运动适应机制是通过有氧训练时肌肉收缩力和收缩速度的结合来实现的。负荷率是控制肺通气量和氧摄取率的过度条件，与急性适应一样。此外，在慢性适应方面，大扭矩运动的LT强度和LT适应速度均高于小扭矩运动。低力矩运动时，基础负荷与饱和负荷的功率差大于低力矩运动时的功率差。

The purpose of this study is to examine the derivation of voluntarily molecular biological adaptability such as respiratory and circulatory system, skeletal muscle about mechanism for acute and chronic exercise induced adaptability by combining force and velocity of muscle contraction at aerobic training.Our result showed that high torque exercise at LT work-load rate was the excess condition for controlling pulmonary ventilation and oxygen intake rate as for acute adaptation. In addition, as for chronic adaptation, LT intensity and its adaptation for high torque exercise were higher and faster than those for low torque exercise. The difference of LT watts at baseline and saturated level was higher than that for low torque exercise.