A factor and a study about the relations with the nursing care to cause surgical site infection
导致手术部位感染的因素及与护理的关系研究
基本信息
- 批准号:19592442
- 负责人:
- 金额:$ 2.83万
- 依托单位:
- 依托单位国家:日本
- 项目类别:Grant-in-Aid for Scientific Research (C)
- 财政年份:2007
- 资助国家:日本
- 起止时间:2007 至 2009
- 项目状态:已结题
- 来源:
- 关键词:
项目摘要
I let you extend hospitalization, and it is said to decrease the QOL of the patient the outbreak of the surgical site infection, but it is not made clear about the skin care of the surgical site. For 41 cases to perform a digestive organ surgical operation, I used two kinds of wound covering materials. And I investigated a physiology and the bacteria outbreak of the skin in the wound covering material pasting part. There was little SSI outbreak and, by this investigation, was not able to confirm the difference. However, the wound part of A group had much outbreak of CNS. A skin physiology was one item of the quantity of corneum substance water, and A group showed the price that was significantly higher than B group. A clot was easy to be made than the above-mentioned result in A group, and the possibility that became the groundwork of the bacteria outbreak was suggested. About the intervention of the nursing care,Skin care I fix the quantity of water of the skin after an operation in A group for a wound part and to remove transient bacteria is important. In addition, in B group, intervention of the skin care except transient bacteria such as the Pseudomonas aeruginosa was suggested.
我让您延长住院治疗,据说可以减少患者的QoL外科现场感染的爆发,但尚不清楚手术部位的皮肤护理。对于41例进行消化器官手术手术的病例,我使用了两种覆盖材料的伤口。我研究了伤口覆盖材料粘贴部分的生理学和细菌爆发。 SSI爆发很少,并且通过这项调查,无法确认差异。但是,一个小组的伤口部分爆发了中枢神经系统。皮肤生理学是角膜水水数量的一项,一组显示的价格明显高于B组。比上述结果很容易制成一个凝块,并提出了成为细菌爆发的基础的可能性。关于护理护理的干预,皮肤护理我在组中手术后修复皮肤的水数,并去除瞬态细菌很重要。此外,在B组中,建议除瞬时细菌(例如铜绿假单胞菌)以外的皮肤护理干预。
项目成果
期刊论文数量(0)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)
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NISHIO Naomi其他文献
The expression of GADD34is induced by various TLR kligands and affect the cytokine productions via TLR signaling
GADD34 的表达由多种 TLR K 配体诱导,并通过 TLR 信号转导影响细胞因子的产生
- DOI:
- 发表时间:
2012 - 期刊:
- 影响因子:0
- 作者:
ITO Sachiko;NISHIO Naomi;TANAKA Yuriko;ISOBE Ken-ichi - 通讯作者:
ISOBE Ken-ichi
The expression of GADD34 is inducesd by various TLR ligands and affect the cytokine productions via TLR signaling
GADD34 的表达由多种 TLR 配体诱导,并通过 TLR 信号转导影响细胞因子的产生
- DOI:
- 发表时间:
2012 - 期刊:
- 影响因子:0
- 作者:
ITO Sachiko;NISHIO Naomi;TANAKA Yuriko; ISOBE Ken-ichi - 通讯作者:
ISOBE Ken-ichi
Loss of GADD34 induces early immuneaging : impact on the proliferation of neutrophil-lineage cells
GADD34 的缺失会诱导早期免疫衰老:对中性粒细胞谱系细胞增殖的影响
- DOI:
- 发表时间:
2012 - 期刊:
- 影响因子:0
- 作者:
NISHIO Naomi;ITO Sachiko;ISOBE Ken-ichi - 通讯作者:
ISOBE Ken-ichi
Loss of GADD34 indues early immune aging: impact on the proliferation of neutrophil-lineage cells
GADD34 缺失导致早期免疫衰老:对中性粒细胞谱系细胞增殖的影响
- DOI:
- 发表时间:
2012 - 期刊:
- 影响因子:0
- 作者:
NISHIO Naomi;ITO Sachiko;ISOBE Ken-ichi - 通讯作者:
ISOBE Ken-ichi
NISHIO Naomi的其他文献
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{{ truncateString('NISHIO Naomi', 18)}}的其他基金
Analysis of GADD34 deficiency effects on obesity-inducetype 2 diabetes and fatty liver development with the aging
GADD34缺乏对肥胖诱发的2型糖尿病和脂肪肝随年龄增长的影响分析
- 批准号:
26870257 - 财政年份:2014
- 资助金额:
$ 2.83万 - 项目类别:
Grant-in-Aid for Young Scientists (B)
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- 批准号:
2109428 - 财政年份:2021
- 资助金额:
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Standard Grant