FASEB Conference: Ion Channel Regulation
FASEB 会议:离子通道调节
基本信息
- 批准号:7005554
- 负责人:
- 金额:$ 1万
- 依托单位:
- 依托单位国家:美国
- 项目类别:
- 财政年份:2005
- 资助国家:美国
- 起止时间:2005-07-15 至 2006-06-30
- 项目状态:已结题
- 来源:
- 关键词:
项目摘要
DESCRIPTION (provided by applicant):
This is a revised application for partial support of the FASEB Summer Research Conference on Ion Channel Regulation (June 4-9, 2005, Snowmass Village, Colorado). Major focus of this Meeting will be on regulation of channel expression, posttranslational modification, trafficking, activity and biological function by a variety of signaling mechanisms, involving G proteins, second messengers, protein phosphorylation, cytoskeleton, membrane lipids and other. Novel signaling pathways and chanellopathies will be discussed. All the invited speakers are confirmed: they represent prominent scientists, including 6 female and 8 junior investigators, who have made important discoveries in the field of ion channel regulation. Many recent advances have occurred in ion channel regulation research, making this an important conference topic in its own right. The FASEB Conference on Ion Channel Regulation is a biennial meeting that alternates with a Gordon Conference on Ion Channels, and is designed to satisfy the growing need in significant expansion of the field and filling the gaps between channel structure and complex biological regulation, with the emphasis on signaling cascades and physiological functions. It is designed to be integrative and interdisciplinary in nature, bringing together investigators from biophysics, biochemistry, cell and molecular biology, physiology, neuroscience and medicine. This conference will highlight exciting discoveries of new families of ion channels that are regulated by novel mechanisms and serve novel biological and pathophysiological functions. More than 120 participants are expected, including pre-and postdoctoral trainees, young faculty, female and minority investigators. To promote participation of women, young investigators, and other underrepresented groups, Conference Fellowships will be offered, and they will be encouraged to present short talks that will complement each session. Requested funds will be used to provide Conference Fellowships for under-represented groups, and to partially cover travel expenses for the speakers from the U.S.
描述(由申请人提供):
项目成果
期刊论文数量(0)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)
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Diomedes E. Logothetis其他文献
Modulation of a GIRK1 Active Mutant Subunit by Protein Kinase C Isoforms
- DOI:
10.1016/j.bpj.2019.11.2360 - 发表时间:
2020-02-07 - 期刊:
- 影响因子:
- 作者:
Aishwarya Chandrashekar;Kirin Gada;Yu Xu;Takeharu Kawano;Leigh D. Plant;Diomedes E. Logothetis - 通讯作者:
Diomedes E. Logothetis
Regulation of CFTR by the membrane phospholipid PIP2
- DOI:
10.1016/j.bpj.2023.11.2458 - 发表时间:
2024-02-08 - 期刊:
- 影响因子:
- 作者:
Ioanna Maria Vynichaki;Laszlo Csanady;Diomedes E. Logothetis - 通讯作者:
Diomedes E. Logothetis
Cooperative Regulation of Slack Channel by Na<sup>+</sup>, Cl<sup>−</sup> and PIP2
- DOI:
10.1016/j.bpj.2011.11.753 - 发表时间:
2012-01-31 - 期刊:
- 影响因子:
- 作者:
Zhe Zhang;Qiongyao Tang;Diomedes E. Logothetis - 通讯作者:
Diomedes E. Logothetis
Functional Relevance of Orthosteric Binding Site of 5-Hydroxytryptamine 2A Receptor and the Mechanism of Receptor Activation
- DOI:
10.1016/j.bpj.2019.11.671 - 发表时间:
2020-02-07 - 期刊:
- 影响因子:
- 作者:
Yu Xu;Guoqing Xiang;Takeharu Kawano;Diomedes E. Logothetis - 通讯作者:
Diomedes E. Logothetis
Hypercholesterolemia Induces Upregulation of K<sub>ACh</sub> Cardiac Currents
- DOI:
10.1016/j.bpj.2011.11.1664 - 发表时间:
2012-01-31 - 期刊:
- 影响因子:
- 作者:
Wu Deng;Anna N. Bukiya;Aldo A. Rodríguez-Menchaca;Zhe Zhang;Clive M. Baumgarten;Diomedes E. Logothetis;Irena Levitan;Avia Rosenhouse-Dantsker - 通讯作者:
Avia Rosenhouse-Dantsker
Diomedes E. Logothetis的其他文献
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{{ truncateString('Diomedes E. Logothetis', 18)}}的其他基金
Dravet Syndrome Anti-Epileptic Control by Targeting GIRK Channels
通过针对 GIRK 通道进行 Dravet 综合征抗癫痫控制
- 批准号:
10638439 - 财政年份:2023
- 资助金额:
$ 1万 - 项目类别:
FUNCTIONALLY IMPORTANT PKA PHOSPHORYLATION SITE IN A KIR3 CHANNEL SUBUNIT
KIR3 通道亚基中功能重要的 PKA 磷酸化位点
- 批准号:
8361551 - 财政年份:2011
- 资助金额:
$ 1万 - 项目类别:
FUNCTIONALLY IMPORTANT PKA PHOSPHORYLATION SITE IN A KIR3 CHANNEL SUBUNIT
KIR3 通道亚基中功能重要的 PKA 磷酸化位点
- 批准号:
8169180 - 财政年份:2010
- 资助金额:
$ 1万 - 项目类别:
Modulation of Kir Channel Function by Phosphorylation
通过磷酸化调节 Kir 通道功能
- 批准号:
7806531 - 财政年份:2009
- 资助金额:
$ 1万 - 项目类别:
Modulation of Kir Channel Function by Phosphorylation
通过磷酸化调节 Kir 通道功能
- 批准号:
8055306 - 财政年份:2009
- 资助金额:
$ 1万 - 项目类别:
Modulation of Kir Channel Function by Phosphorylation
通过磷酸化调节 Kir 通道功能
- 批准号:
7653214 - 财政年份:2009
- 资助金额:
$ 1万 - 项目类别:
FUNCTIONALLY IMPORTANT PKA PHOSPHORYLATION SITE IN A KIR3 CHANNEL SUBUNIT
KIR3 通道亚基中功能重要的 PKA 磷酸化位点
- 批准号:
7954149 - 财政年份:2009
- 资助金额:
$ 1万 - 项目类别:
Modulation of Kir Channel Function by Phosphorylation
通过磷酸化调节 Kir 通道功能
- 批准号:
8239544 - 财政年份:2009
- 资助金额:
$ 1万 - 项目类别:
Protein kinase C-dependent inhibition of Kir channels
Kir 通道的蛋白激酶 C 依赖性抑制
- 批准号:
6752128 - 财政年份:2003
- 资助金额:
$ 1万 - 项目类别:
Protein kinase C-dependent inhibition of Kir channels
Kir 通道的蛋白激酶 C 依赖性抑制
- 批准号:
6947290 - 财政年份:2003
- 资助金额:
$ 1万 - 项目类别:
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