Physiologic Functions of NF-kB Signaling in Intestinal Epithelium

肠上皮中 NF-kB 信号传导的生理功能

• 批准号: 7425090
• 负责人: Martin Frederick KAGNOFF
• 金额: $ 27.4万
• 依托单位: UNIVERSITY OF CALIFORNIA, SAN DIEGO
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-04-01 至 2008-03-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7425090
• 关键词: Affect; Apoptosis; Apoptotic; Celiac Disease; Cell Survival; Cell physiology; Chemical Injury; Communication; Data; Defense Mechanisms; Enteral; Environment; Epithelial; Epithelial Cells; Essential Genes; Event; Funding; Gamma Rays; Gene Targeting; Generations; Genes; Genetic Recombination; Genetic Transcription; Goals; Host Defense; Human; I-kappa B Proteins; Immune; Immune response; In Vitro; Indium; Infection; Inflammatory Bowel Diseases; Inflammatory Response; Injury; Intestinal Mucosa; Intestines; Ionizing radiation; Knock-out; Lead; Link; Mediator of activation protein; Microbe; Modeling; Mucositis; Mus; NF-kappa B; Natural Immunity; Pathway interactions; Phosphotransferases; Physiological; Play; Principal Investigator; Process; Production; Regulation; Research; Role; Signal Pathway; Signal Transduction; Site; Surface; System; Testing; Wound Healing; Wounds and Injuries; acquired immunity; base; cell injury; cell motility; crypt cell; design; environmental agent; in vitro Model; in vivo; intestinal epithelium; microbial; novel; pathogen; programs; promoter; recombinase; response; seal; transcription factor; villin; wound