Adenovirus Activation of Antigen Presenting Cells Through DNA Sensing Mechanisms

腺病毒通过 DNA 传感机制激活抗原呈递细胞

• 批准号: 8105569
• 负责人: ERIK S FALCK-PEDERSEN
• 金额: $ 50.78万
• 依托单位: WEILL MEDICAL COLL OF CORNELL UNIV
• 依托单位国家: 美国
• 财政年份: 2004
• 资助国家: 美国
• 起止时间: 2004-12-01 至 2011-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8105569
• 关键词: Address; Adenovirus Infections; Adenovirus Vector; Adenoviruses; Adjuvant; Affect; Antigen-Presenting Cells; Antiviral Agents; Antiviral Response; Area; Autocrine Communication; Autoimmune Diseases; Biological Models; Biology; Bone Marrow; Capsid; Cell Line; Cell Maturation; Cell membrane; Cells; Complement; DNA; DNA Vaccines; Dendritic Cells; Disease; Event; Exposure to; Funding; Gene Transfer; Hepatocyte; IRF3 gene; Immune; Immune System Diseases; Immune Targeting; Immune response; Immune system; Immunity; In Vitro; Infection; Inflammatory; Integrins; Interferons; Intrinsic factor; Knowledge; Ligation; Mediator of activation protein; Modeling; Modification; Molecular; Mus; Mutation; Nature; Outcome; Pathway interactions; Progress Reports; Proteins; Protocols documentation; Recombinants; Research; Role; Sentinel; Signal Transduction; Staging; Sting Injury; System; T-Lymphocyte; TBK1 gene; TNF gene; Therapeutic; Translating; Vaccines; Viral; Virus Diseases; adaptive immunity; adenovirus penton protein; anti-cancer therapeutic; arm; autocrine; base; biological adaptation to stress; chemokine; cytokine; gene therapy; gene transfer vector; immune activation; immune function; in vitro Model; in vivo; macrophage; migration; paracrine; pathogen; public health relevance; research study; response; sensor; type I interferon receptor; viral DNA; virus host interaction