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Mechanisms of coronary endothelial dysfunction in diabetes

Mechanisms of coronary endothelial dysfunction in diabetes
糖尿病冠状动脉内皮功能障碍的机制
批准号：
8227960
负责人：
Ayako Makino
金额：
$12.47万
依托单位：
UNIVERSITY OF ILLINOIS AT CHICAGO
依托单位国家：
美国
项目类别：
财政年份：
2009
资助国家：
美国
项目状态：
已结题
起止时间：
2009-04-01 至 2012-03-31
来源：
https://reporter.nih.gov/project-details/8227960
关键词：
AccountingAttenuatedAwardBasic ScienceBlood VesselsBlood capillariesCardiacCardiovascular systemCell CommunicationCellsChronicComplicationConnexin 43ConnexinsCoronaryCoronary arteryDataDevelopmentDiabetes MellitusDiabetic mouseDown-RegulationEndothelial CellsFunctional disorderGap JunctionsGene ExpressionGenesGoalsHealthHeartHeart failureHyperglycemiaHyperlipidemiaHypertensionLeadLearningLinkMaintenanceMentorshipMolecularMorbidity - disease rateMuscle CellsPathogenesisPathway interactionsPlayProstaglandinsRegulationResearch PersonnelResearch Project GrantsResistanceRiskRisk FactorsRoleTestingTherapeuticTransgenic MiceTranslational ResearchVascular DiseasesVascular Endothelium-Dependent RelaxationVascular blood supplyVascular resistanceWorkbasecapillarydensitydiabeticdiabetic patientglycosylationinsightintercellular communicationmortalitymouse modelnovel therapeutic interventionoverexpressionpeptide O-linked N-acetylglucosamine-beta-N-acetylglucosaminidasepreventprotein expressiontranscription factor

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期刊论文(1)
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科研奖励(0)
会议论文
Mitochondrial function in vascular endothelial cell in diabetes.
DOI： 10.1540/jsmr.48.1
发表时间： 2012
期刊： Journal of smooth muscle research = Nihon Heikatsukin Gakkai kikanshi
影响因子： --
作者： [Pangare M, Makino A]
通讯作者： Makino A
Role of Gap Junction in Hypoxia-induced Pulmonary Hypertension
Role of Gap Junction in Hypoxia-induced Pulmonary Hypertension
Cellular and Molecular Mechanisms of Coronary Endothelial Dysfunction in Diabetes
  • 批准号：
    8688345
  • 项目类别：
  • 资助金额：
    $37.36万
  • 财政年份：
    2012
  • 负责人：
    Ayako Makino
  • 依托单位：
    UNIVERSITY OF ARIZONA
Cellular and Molecular Mechanisms of Coronary Endothelial Dysfunction in Diabetes
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