MECHANISM OF REPLICATION TERMINATION

复制终止机制

• 批准号: 7957845
• 负责人: DEEPAK BASTIA
• 金额: $ 0.33万
• 依托单位: UNIVERSITY OF WASHINGTON
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-09-01 至 2010-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7957845
• 关键词: Biology; Computer Retrieval of Information on Scientific Projects Database; DNA biosynthesis; Funding; Fungal Genome; Genetic Recombination; Genome Stability; Grant; Institution; Process; Regulation; Research; Research Personnel; Resources; Source; United States National Institutes of Health; clinically significant

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. DNA replication is an essential process and its regulation has fundamental and clinical significance. In this context Replication termination is highly important because it regulates replication, connects it with recombination and confirms genome stability.

这个子项目是许多研究子项目中的一个 由NIH/NCRR资助的中心赠款提供的资源。子项目和 研究者（PI）可能从另一个NIH来源获得了主要资金， 因此可以在其他CRISP条目中表示。所列机构为 研究中心，而研究中心不一定是研究者所在的机构。 DNA复制是一个重要的生物学过程，其调控具有重要的基础和临床意义。在这种情况下，复制终止是非常重要的，因为它调节复制，将其与重组联系起来，并确认基因组的稳定性。