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MECHANISM OF REPLICATION TERMINATION

MECHANISM OF REPLICATION TERMINATION
复制终止机制
批准号：
8171240
负责人：
DEEPAK BASTIA
金额：
$0.24万
依托单位：
UNIVERSITY OF WASHINGTON
依托单位国家：
美国
项目类别：
财政年份：
2010
资助国家：
美国
项目状态：
已结题
起止时间：
2010-09-01 至 2011-08-31
来源：
https://reporter.nih.gov/project-details/8171240
关键词：
Computer Retrieval of Information on Scientific Projects DatabaseDNA biosynthesisFundingGenetic RecombinationGenome StabilityGrantInstitutionProcessRegulationResearchResearch PersonnelResourcesSourceUnited States National Institutes of Healthclinically significant

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中文摘要
翻译
该子项目是利用 由NIH/NCRR资助的中心赠款提供的资源。子项目和 研究者（PI）可能从另一个NIH来源获得主要资金， 因此可以在其他CRISP条目中表示。列出的机构是 中心，不一定是研究者的机构。 DNA复制是一个重要的生物学过程，其调控具有重要的基础和临床意义。在这种情况下，复制终止是非常重要的，因为它调节复制，将其与重组联系起来，并确认基因组的稳定性。
英文摘要
This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. DNA replication is an essential process and its regulation has fundamental and clinical significance. In this context Replication termination is highly important because it regulates replication, connects it with recombination and confirms genome stability.
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Control of Programmed Replication fork Arrest by Chromosome Kissing
Control of Programmed Replication fork Arrest by Chromosome Kissing
Control of Programmed Replication fork Arrest by Chromosome Kissing
Control of Programmed Replication fork Arrest by Chromosome Kissing
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