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Mechanisms of Retinal Vascular Permeability in Diabetes

Mechanisms of Retinal Vascular Permeability in Diabetes
糖尿病视网膜血管通透性的机制
批准号：
8206694
负责人：
David Antonetti
金额：
$36.65万
依托单位：
UNIVERSITY OF MICHIGAN AT ANN ARBOR
依托单位国家：
美国
项目类别：
财政年份：
1998
资助国家：
美国
项目状态：
已结题
起止时间：
1998-09-01 至 2014-04-30
来源：
https://reporter.nih.gov/project-details/8206694
关键词：
Advanced Glycosylation End ProductsAlanineBiochemicalBlindnessBlood VesselsBlood-Retinal BarrierCCL2 geneCattleCell DeathCell ProliferationCell divisionCell membraneCellsComplexCytoplasmDataDevelopmentDiabetes MellitusDiabetic RetinopathyDiseaseDoseElementsEndocytosisEndosomesEndothelial CellsEpithelialFDA approvedFigs - dietaryFunctional disorderFundingGene DeletionGene ExpressionGoalsGolgi ApparatusGrowth FactorHealthHyperglycemiaHyperlipidemiaHyperplasiaImmigrationInflammatoryIntegral Membrane ProteinInterleukin-1LaboratoriesLeadLifeLinkLiteratureMass Spectrum AnalysisMeasuresMedicalMetabolicMolecularMusMutateMutationOxygenPathogenesisPathway interactionsPermeabilityPhenotypePhosphorylationPhosphorylation SiteProliferatingProtein IsoformsProtein Kinase CProteinsPublishingRattusReceptor GeneRegulationResearchRetinaRetinalRetinal DiseasesRoleSignal PathwaySignal TransductionSiteSmall Interfering RNAStomachStructure of retinal pigment epitheliumTestingTherapeuticTherapeutic InterventionTight JunctionsTimeTubeTumor Necrosis Factor ReceptorTumor Necrosis Factor-alphaUbiquitinationUnited StatesVascular Endothelial CellVascular Endothelial Growth FactorsVascular Permeabilitiesangiogenesisatypical protein kinase Cbasecell growthcellular imagingcomparativecytokineepsinimmunocytochemistryin vivoinhibitor/antagonistmacular edemamutantnew therapeutic targetnovelnovel therapeuticsoccludinoverexpressionpreventprotein kinase C betaresearch studyresponsetherapeutic targettrafficking

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