PATHOGENESIS OF EBV INFECTION IN THE RHESUS MACAQUE ANIMAL MODEL

恒河猴动物模型中 EBV 感染的发病机制

• 批准号: 8172798
• 负责人: Frederick C. Wang
• 金额: $ 20.24万
• 依托单位: HARVARD MEDICAL SCHOOL
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-05-01 至 2011-04-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8172798
• 关键词: Animal Model; Computer Retrieval of Information on Scientific Projects Database; Epstein-Barr Virus Infections; Funding; Grant; Immune; Immune response; Infection; Institution; Lymphocryptovirus; Macaca mulatta; Pathogenesis; Research; Research Personnel; Resources; Source; United States National Institutes of Health; base; latent persistent infection

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Rhesus lymphocryptovirus (RhLCV) is genetically similar to EBV, and results in persistent latent infection in rhesus macaques. In order to investigate the immune basis of latency induced by EBV infection, rhLCV-specific immune responses are being evaluated in rhesus macaques. Aids Related

这个子项目是许多利用 由NIH/NCRR资助的中心赠款提供的资源。子项目和 研究者（PI）可能从另一个NIH来源获得了主要资金， 因此可以在其他CRISP条目中表示。所列机构为 研究中心，而研究中心不一定是研究者所在的机构。 恒河猴淋巴隐病毒（Rhesus lymphocyptovirus，RhLCV）与EB病毒在基因上相似，可引起恒河猴持续性潜伏感染。 为了研究EBV感染诱导的潜伏期的免疫基础，正在恒河猴中评价rhLCV特异性免疫应答。 艾滋病相关