ROS Modulation of Innate and Adaptive Immunity in RA

ROS 对 RA 先天性和适应性免疫的调节

• 批准号: 8309519
• 负责人: John D Mountz
• 金额: $ 5.4万
• 依托单位: UNIVERSITY OF ALABAMA AT BIRMINGHAM
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-09-01 至 2012-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8309519
• 关键词: Affect; Antibodies; Antigens; Antioxidants; Autoimmune Diseases; Autoimmune Process; B-Lymphocytes; Cattle; Cells; Chronic; Collagen; Collagen Arthritis; Collagen Type II; DBA/1 Mouse; Dendritic Cells; Disease; Equilibrium; Fibroblasts; Generations; Immune; Immunization; Immunohistochemistry; Immunotherapeutic agent; Inflammation; Inflammatory; Insulin-Dependent Diabetes Mellitus; Interferons; Interleukin-10; Interleukin-17; Interleukin-4; Interleukin-6; Joints; Mediating; Modeling; Mus; Natural Immunity; Oxidation-Reduction; Pathogenesis; Production; Reactive Oxygen Species; Research Proposals; Rheumatism; Rheumatoid Arthritis; Rodent Model; Role; Signal Pathway; Signal Transduction; Staining method; Stains; Swelling; Synovial Membrane; T-Lymphocyte; TNF gene; adaptive immunity; autoreactive T cell; cytokine; immune activation; joint destruction; macrophage; novel; prevent; response