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mt-Nd2 and resistance to autoimmune diabetes

mt-Nd2 and resistance to autoimmune diabetes
mt-Nd2 与自身免疫性糖尿病抵抗力
批准号：
8331716
负责人：
CLAYTON E MATHEWS
金额：
$10.69万
依托单位：
UNIVERSITY OF FLORIDA
依托单位国家：
美国
项目类别：
财政年份：
2006
资助国家：
美国
项目状态：
已结题
起止时间：
2006-04-01 至 2012-05-31
来源：
https://reporter.nih.gov/project-details/8331716
关键词：
ALR MouseAccountingAllelesAlloxanAmino AcidsAntigen TargetingApoptoticAttentionAutoimmune DiabetesAutoimmune ProcessBeta CellBiological AssayBlood PressureBreedingCD8-Positive T-LymphocytesCandidate Disease GeneCatalytic DomainCell DeathCell LineCell NucleusCell physiologyCell-Mediated CytolysisCellsCessation of lifeCollectionCommunicationComplexCytochrome c ReductaseCytolysisCytotoxic T-LymphocytesDataDevelopmentDiabetes MellitusEngraftmentFigs - dietaryFutureGenerationsGenesGeneticGenetic PolymorphismGenetic Predisposition to DiseaseGenetic VariationGlucoseGoalsGranzymeHaplotypesHumanHuman CharacteristicsHybrid CellsHybridsImmuneImmune systemIn VitroInbred NOD MiceIndividualInjuryInsulinInsulin-Dependent Diabetes MellitusIslets of LangerhansIslets of Langerhans TransplantationKnowledgeLeadLeucineLifeLigationLightLinkLongevityMediatingMembrane PotentialsMetabolismMitochondriaMitochondrial DNAMouse StrainsMusMutationMyocardial InfarctionMyotonic DystrophyNon-Insulin-Dependent Diabetes MellitusNuclearOxidative StressPathogenesisPathologyPathway interactionsPatientsPeptidesPlayPopulationPredispositionProductionProtein KinaseProtocols documentationReactive Oxygen SpeciesReportingResearchResearch DesignResistanceRiskRoleSignal TransductionSingle Nucleotide PolymorphismSourceStressStructure of beta Cell of isletT-LymphocyteTNF geneTNFRSF1A geneTestingTissuesTransplantationTumor Necrosis Factor Ligand Superfamily Member 6Tumor Necrosis Factor ReceptorVariantVisualWorkatherogenesiscellular engineeringcohortcytokinefree radical oxygenfunctional disabilitygenetic analysisgenetic elementgenetic pedigreegenetic variantglucose toleranceglucose-6-phosphataseimprovedinsightinsulin secretionisletkillingsmanmembermitochondrial DNA mutationmouse modelperforinpreventreceptorstemward

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Discovery and Roles of In Situ Islet Neoantigens in Human Type 1 Diabetes
  • 批准号：
    10589578
  • 项目类别：
  • 资助金额：
    $71.57万
  • 财政年份：
    2023
  • 负责人：
    CLAYTON E MATHEWS
  • 依托单位：
    UNIVERSITY OF FLORIDA
Determining the mechanism of IFIH1 disease-associated variants on beta-cell and immune responses in Type 1 diabetes
Determining the mechanism of IFIH1 disease-associated variants on beta-cell and immune responses in Type 1 diabetes
Organ Specific Project
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