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DAP12 and ITAM-signals in Osteoclastogenesis

DAP12 and ITAM-signals in Osteoclastogenesis
破骨细胞生成中的 DAP12 和 ITAM 信号
批准号：
7906050
负责人：
Mary C Nakamura
金额：
$0.0万
依托单位：
VETERANS AFFAIRS MED CTR SAN FRANCISCO
依托单位国家：
美国
项目类别：
财政年份：
2009
资助国家：
美国
项目状态：
已结题
起止时间：
2009-10-01 至 2014-03-31
来源：
https://reporter.nih.gov/project-details/7906050
关键词：
AgingAntioxidantsAreaBone ResorptionBone remodelingCalciumCell Surface ReceptorsCellsChronic DiseaseDefectDegenerative polyarthritisDevelopmentDietDiseaseEquilibriumEstrogensFeedbackFractureGenesHematopoieticHip FracturesITAMIn VitroIncidenceInflammatoryLeadLifeLinkLipopolysaccharidesMacrophage Colony-Stimulating FactorMacrophage Colony-Stimulating Factor ReceptorMalignant NeoplasmsMediatingMetastatic Neoplasm to the BoneMorbidity - disease rateMusMyelogenousMyeloid CellsNF-kappa BNeoplasm MetastasisOrganOsteoblastsOsteoclastsOsteogenesisOsteoporosisPathologicPatientsPhenotypePhosphorylationProductionProtein Tyrosine KinaseProteinsReactive Oxygen SpeciesReceptor ActivationReceptor SignalingResearchRheumatoid ArthritisSignal PathwaySignal TransductionSpinal FracturesStimulusTEC Protein Tyrosine KinaseTNFSF11 geneTYROBP geneWomanadapter proteinbasebonebone lossc-fms Proto-Oncogenescell typehuman diseasein vivointerestmacrophagemenmonocytemortalitynovel therapeuticsosteoclastogenesisosteoporosis with pathological fracturepatient populationprecursor cellpreventpublic health relevancereceptorresponsetranscription factor

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