The Role of Beta-Gal Mediated Interactions in Prostate Cancer Metastasis

β-半乳糖介导的相互作用在前列腺癌转移中的作用

• 批准号: 7910715
• 负责人: VLADISLAV V GLINSKII
• 金额: --
• 依托单位: HARRY S. TRUMAN MEMORIAL VA HOSPITAL
• 依托单位国家: 美国
• 财政年份: 2009
• 资助国家: 美国
• 起止时间: 2009-10-01 至 2013-12-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7910715
• 关键词: Adhesives; Affect; Aging; Apoptosis; Apoptotic; Atomic Force Microscopy; Binding Proteins; Bone Diseases; Carbohydrates; Carcinoma; Caspase; Cell Adhesion; Cell Aggregation; Cell Communication; Cell surface; Cells; Cellular biology; Cessation of life; Chemotherapy-Oncologic Procedure; Citrus; Combined Modality Therapy; Cytotoxic agent; Data; Development; Disaccharides; Disease; Disease Progression; Disseminated Malignant Neoplasm; Distant; Drug effect disorder; Effectiveness; Endothelial Cells; Endothelium; Event; Family; Galactosides; Galectin 2; Galectin 3; Goals; Growth; Health; Homeostasis; Human; Image; In Vitro; Injection of therapeutic agent; Integrin alpha3beta1; Integrins; Intractable Pain; Leucine; Luciferases; Malignant Epithelial Cell; Malignant Neoplasms; Malignant neoplasm of prostate; Maps; Mediating; Metastatic Neoplasm to the Bone; Metastatic Prostate Cancer; Mitochondria; Modality; Modeling; Molecular; Monitor; Morbidity - disease rate; Mucins; Neoplasm Metastasis; Organ; Pathological fracture; Pathway interactions; Patients; Pectins; Phosphorylation; Play; Population; Pre-Clinical Model; Predisposition; Process; Property; Prostate; Prostate carcinoma; Prostatic Diseases; Protein Dynamics; Quality of life; Regimen; Regulation; Relative (related person); Research; Role; Signal Pathway; Skeleton; Techniques; Testing; Thompson-Friedenreich Antigen; Time; TimeLine; Translating; Tumor Burden; Veterans; adhesion process; base; beta-galactoside; bone; cancer cell; cancer diagnosis; cell motility; chemotherapy; clinical practice; docetaxel; in vivo; inhibitor/antagonist; male; men; mortality; mouse model; neoplastic cell; new therapeutic target; novel; novel strategies; novel therapeutic intervention; prevent; research study; spinal cord compression; src-Family Kinases; therapeutic target; tumor