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Impact of genetic variation TLR/CD14 pathways and smoking in RA

Impact of genetic variation TLR/CD14 pathways and smoking in RA
遗传变异 TLR/CD14 通路和吸烟对 RA 的影响
批准号：
7787500
负责人：
TED RICHARD MIKULS
金额：
$0.0万
依托单位：
OMAHA VA MEDICAL CENTER
依托单位国家：
美国
项目类别：
财政年份：
2009
资助国家：
美国
项目状态：
已结题
起止时间：
2009-04-01 至 2012-06-30
来源：
https://reporter.nih.gov/project-details/7787500
关键词：
AffectAfrican AmericanAgingAlabamaAllelesAllyAmericanAnimal ModelAntigensArthritisAttenuatedAutoantibodiesAutoimmune ProcessAutoimmunityCD14 geneCandidate Disease GeneCaucasiansCaucasoid RaceCellsCollaborationsDataDetectionDiseaseDisease susceptibilityEndotoxinsEpitopesEvaluationExposure toFibrinogenGene-ModifiedGeneral PopulationGenesGeneticGenetic PolymorphismGenetic VariationHLA-DRB1HealthHealth Care CostsHealthcareHeat shock proteinsHigh PrevalenceInflammatoryLigandsLinkLungLung noduleMeasuresMediatingMediator of activation proteinMinorityMorbidity - disease rateNatural ImmunityOutcomePTPN22 genePathologyPathway interactionsPattern recognition receptorPeptide antibodiesPlayPopulationPrevalenceProductionProtein Tyrosine PhosphataseReceptor GeneReceptor SignalingResearchResearch PersonnelRheumatoid ArthritisRheumatoid FactorRheumatoid NoduleRiskRisk FactorsRoleSerumSeveritiesSeverity of illnessSingle Nucleotide PolymorphismSmokerSmokingSynovial MembraneTLR9 geneTechniquesTestingToll-Like Receptor 1Toll-Like Receptor 2Toll-Like Receptor PathwayToll-like receptor 6Toll-like receptorsUniversitiesVariantVeteransWomanadaptive immunityarthritis registrycigarette smokingcigarette smokingcohortcyclic citrullinated peptidecytokinedisabilitydisease phenotypegenetic risk factorhuman TLR7 proteininsightjoint destructionmalemenmortalitynovelpublic health relevancereceptor expressionsmoking prevalencetoll-like receptor 4

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Professional Development Core
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