植物-病原菌互作过程中的植物过敏反应现象，一直是植物病理学家关心的问题，其中细胞快速程序性死亡的精确调控机制、关键通路尚有许多未解之谜，有待深入研究。本课题组通过比较植物细胞快速程序性死亡与动物细胞凋亡机制的差异，并结合本实验室的研究积累，提出植物-病原菌互作过程中植物抗氰呼吸是过敏反应节点的假说，并拟以小麦锈菌诱导的小麦过敏反应、水稻白叶枯病菌诱导的水稻过敏反应为实验体系，研究这一假说的分子基础。相信通过本项目的研究，能够部分阐明病原菌诱导的植物过敏反应中细胞快速程序性死亡的精确调控机制及关键通路，初步揭示植物-病原菌互作过程中植物抗氰呼吸成为过敏反应节点的分子基础。

Plant - pathogen interaction in the process of plant allergic reactions, has been a concern of plant pathologists, including rapid regulation of cell death mechanism, the key path there are many unsolved mysteries to be further studied.In this study, we compared the rapid apoptosis of plant cells and the difference of animal cell apoptosis mechanism, and combined with the accumulation of laboratory research, the plant-pathogen interaction process of plant cyanide-resistant respiration is the hypersensitivity node hypothesis, and to the wheat allergic reaction induced by Puccinia striiformis West and the rice allergic reaction induced by Xanthomonas oryzae pv.oryzae (Ishiyama)were used as experimental system to study the molecular basis of this hypothesis.Through the study of this project, it is possible to clarify the precise regulation mechanism and key pathways of rapid programmed cell death in plant allergic reactions induced by pathogens.As far as possible to reveal the plant-pathogen interaction process of plant cyanide-resistant respiratory allergic reaction node molecular basis.