Effect of Bite Raising Exerting Neuro-Musclar Mechanism for Musticatory Muscles

咬痕作为一种保存疗法被广泛应用于临床，在目前口腔颌面功能障碍患者增多的情况下，其临床意义变得更加重要。然而，关于咬痕咬合力的具体指标仍然不清楚，甚至连咬合力上升的生理机制也不是很清楚。一开始，我们重点研究咬合，认为咬合在口腔颌面功能障碍的发生发展中起着致病作用，尤其是咬合后咬合力调节机制的动态特性。实验对象为6名牙列保存完好的健康受试者。比较咬合和不咬合后学习的参考咬合力。结果，观察到了一种长期持久的影响，可能与强直、疼痛或咀嚼肌肉的其他功能障碍直接相关。此外，还阐明了咬合反弹对这一效应的影响对于研究咬合指纹的作用机制是重要的。今后，我们将考虑与肌肉长度变化相关的力学因素以及伴随咬合反弹而对运动神经元的输入机制所涉及的额外问题。

Clinical implication of bitesprint that is used widely as a preservation therapy, in the present condition that the stomatognathic dysfunction patients are increasing, has become further important. However, a distinct guideline concerning the amount of bite raising with bitesprint remains not only to be unclear, but also even the physiologic mechanism of its action by raising is not clarified.Our research aimed to analyze an influence of bite rasing to bite force. In the begining, we focused on the clenching, being considered to play a causative role in the development of stomatognathic dysfunction, particularly the dynamic properties of regulatory mechanism of bite force after clenching. The experiments were performed on 6 healthy sujects with well-preserved dentitions. Comparison were made between both learned reference bite forces after clenching and no-clenching. As a result, a long lasting effect that might direct relate to tonus, pain or other dysfunctions of the masticatory muscles was observed. Furthermore it was clarified that the observation of bite rasing influence on this effect is important to investigate the mechanism of action of the bitesprint.In future, we will consider the additional problems involved in the mechanical factors associated with muscle length changing and the inpout mechanism to motor neuron accompanied by bite rasing.