Molecular Genetics of Micrbial Pathogenesis

在我们以小册子的形式详细准备了这个研究组的报告时，我们将我们的结果简要总结如下。其中调控机制表达的微生物发病机制尚不明确或有进展。志贺氏杆菌的侵袭扩散机制。霍乱沙门氏菌的特殊毒力。弧菌的溶血素。绿脓杆菌的碱性蛋白酶。产气荚膜梭菌的气溶酶。粘毛蕨的毛。微生物的致病机制，其活化和/或易位机制是明确的。铜绿假单胞菌的细胞毒素。霍乱弧菌的溶血素。伤寒沙门氏菌的Vi抗原。金黄色葡萄球菌的溶血素和杀白细胞素。粪肠球菌的溶血素和细菌素。肉毒杆菌的C型和E型毒素。其他毒力因素。幽门螺杆菌的脲酶。肠聚集性大肠杆菌对人粘膜的粘附。鸡致病性大肠杆菌的自凝集。牙龈卟啉单胞菌的菌膜。拟杆菌类碎片的神经氨酸酶。质粒R100 TraT产品捐献的血清耐药性。

As we prepared a report of this research group in detail in a booklet form, we summarized our results briefly as follows.Microbial pathogenesis of which regulatory mechanism of expression is clarified or is progress.The invasion-diffusion mechanisms of shigella bacilli. The spv virulence of Salmonella choleraesuis. The hemolysin of Vibrio species. The alkaline-protease of Pseudomonas aerug. The perfringolysin of Clostridium perfringens. The pili of Seratia marcescens.microbial pathogemesis of which activation and/or translocation mechanism is clarified.The cytotoxin of Pseudomonas aeruginosa. The hemolysin of Vibrio cholerae.The Vi antigen of Salmonella typhi. Tge hemolysin and leukocidin of Staphylococcus aureus. The hemolysin and bacteriocin of Enterococcus faecalis. The type C and E toxins of Clostridium botulinum.Other Virulence factors.The urease of Helicobacter pylori. The adherence of enteroaggregative Escherichia coli to human mucosa. The autoagglutination of chicken pathogenic Escherichia coli. The fimbiae of porphyromonas gingivalis. The neuraminidase of Bacteroides frag. The serum resistance donated by plasmid R100 TraT product.