RENAL TUBULAR ACIDOSIS IN PEDIATRIC KIDNEY TRANSPLANT RECIPIENTS

肾小管性酸中毒(RTA)可导致包括生长在内的并发症 衰竭、骨病和肾钙质沉着症。在儿科肾脏中 移植人群中，这些并发症与主要 医疗和心理社会后果。RTA后的发生 移植已经被观察了30年，但其烧灼机制 都没有被很好地理解。这项研究的最终目的是阐明 导致移植后RTA的机制，使它们与 临床病程，并最终确定危险因素。

Renal tubular acidosis (RTA) can lead to complications including growth failure, bone disease, and nephrocalcinosis. In the pediatric renal transplant population, these complications are associated with major medical and psychosocial consequences. The occurence of RTA after transplant has been observed for 30 years, but the causitive mechanisms are not well understood. The ultimate goal of this study is to elucidate the mechanisms causing post-transplant RTA, to correlate them with clinical course, and ultimately to identify risk factors.