GORDON CONFERENCE ON MODIFIERS OF CARDIAC CONTRACTILITY

本次会议将讨论涉及心脏调节的机制， 收缩性 一系列的机制及其修改， 将考虑药理学试剂或生理调节。 的 会议将不仅涉及Ca 2+的分子机制， 跨膜运输，信号传递信息的性质 从质膜到内质网，Ca ~（2+）的调节机制 释放和再摄取，参与调节肌球蛋白或 磷酸二酯酶同工酶和收缩性心肌细胞的分子生物学 蛋白质和膜受体参与调节收缩性，但 也与许多药理学试剂（旧的和新的）， 被认为是调节这些过程的。 希望通过汇集 来自不同学科的活跃科学家，我们将开发一些 了解调节心脏收缩力的机制，以及 深入了解药物改变这些过程的机制。 演讲者的选择是基于科学的卓越性， 个人参与者在该领域的领导力得到证明。 我已要求 仅为排定会议的发言者和主席提供支助。 对其他参与者的支持（约。2/3预期 参与者）将来自个人资金，私人资源和外国 研究机构-我希望！

This conference will deal with mechanisms involved in modulation of cardiac contractility. A host of mechanisms and their modifications by pharmacological agents or physiological regulation will be considered. The conference will deal not only with the molecular mechanisms of Ca2+ transport across the membrane, the nature of signal transmitted information from the surface membrane to SR, the regulation of mechanism of Ca2+ release and re-uptake, the mechanism involved in modulation of myosin or phosphodiestrase isozymes, and the molecular biology of contractile proteins and membrane receptor involved in regulation of contractility, but also with a number of pharmacological agents (old and new) which are thought to modulate these processes. It is hoped that by bringing together active scientists from different disciplines we will develop some understanding of the mechanisms which regulate cardiac contractility, and get some insight into the mechanisms by which drugs alter these processes. The selection of speakers has been based on scientific excellence and proven leadership of the individual participant in the field. I have asked support only for the speakers and chairpersons of the scheduled sessions. Support for the rest of the participants (approx. 2/3 of expected participants) will come from personal funds, private resources and foreign research organizations - I hope!