ENDOTHELIAL PROGENITOR CELLS MOBILIZED BY ARTERIAL INJURY

这个子项目是许多研究子项目中的一个 由NIH/NCRR资助的中心赠款提供的资源。子项目和 研究者（PI）可能从另一个NIH来源获得了主要资金， 因此可在其他CRISP条目中表示。所列机构为 研究中心，而研究中心不一定是研究者所在的机构。 骨髓干细胞（BMSC）响应于缺血性发作（例如心肌梗塞）而动员以修复损伤，或通过细胞因子（例如粒细胞集落刺激因子（G-CSF））动员以用于BMSC收获和自体移植。在目前的研究中，我们调查了G-CSF动员的BMSCs是否与非人灵长类狒狒的缺血诱导修复具有相同的亚型特征。

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Bone marrow stem cells (BMSCs) are mobilized in response to ischemic attacks, e.g. myocardial infarction, to repair the damages, or by cytokines, e.g. granulocyte colony-stimulating factor (G-CSF) for BMSC harvest and autologous transplantation. In the current study, we investigated whether G-CSF-mobilized BMSCs share the same subtype profiles as the ischemia-induced repair in nonhuman primate baboons.