Plant Physiology Increases the Magnitude and Spread of the Transient Climate Response to CO2 in CMIP6 Earth System Models
Plant Physiology Increases the Magnitude and Spread of the Transient Climate Response to CO2 in CMIP6 Earth System Models
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植物生理学增加了 CMIP6 地球系统模型中对二氧化碳的瞬态气候响应的幅度和传播
DOI:
10.1175/jcli-d-20-0078.1
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发表时间:
2020
影响因子:
4.9
通讯作者:
Randerson, James T.
中科院分区:
文献类型:
--
作者:
Zarakas, Claire M.;Swann, Abigail L.;Laguë, Marysa M.;Armour, Kyle C.;Randerson, James T.
Increasing concentrations of CO2in the atmosphere influence climate both through CO2’s role as a greenhouse gas and through its impact on plants. Plants respond to atmospheric CO2concentrations in several ways that can alter surface energy and water fluxes and thus surface climate, including changes in stomatal conductance, water use, and canopy leaf area. These plant physiological responses are already embedded in most Earth system models, and a robust literature demonstrates that they can affect global-scale temperature. However, the physiological contribution to transient warming has yet to be assessed systematically in Earth system models. Here this gap is addressed using carbon cycle simulations from phases 5 and 6 of the Coupled Model Intercomparison Project (CMIP) to isolate the radiative and physiological contributions to the transient climate response (TCR), which is defined as the change in globally averaged near-surface air temperature during the 20-yr window centered on the time of CO2doubling relative to preindustrial CO2concentrations. In CMIP6 models, the physiological effect contributes 0.12°C (σ: 0.09°C; range: 0.02°–0.29°C) of warming to the TCR, corresponding to 6.1% of the full TCR (σ: 3.8%; range: 1.4%–13.9%). Moreover, variation in the physiological contribution to the TCR across models contributes disproportionately more to the intermodel spread of TCR estimates than it does to the mean. The largest contribution of plant physiology to CO2-forced warming—and the intermodel spread in warming—occurs over land, especially in forested regions.
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影响因子:
5.2
作者:
M. Saint‐Lu;R. Chadwick;F. H. Lambert;M. Collins
通讯作者:
M. Collins
影响因子:
5.2
作者:
L. Parsons;M. K. Brennan;Robert C.J. Wills;C. Proistosescu;C. Proistosescu
通讯作者:
L. Parsons;M. K. Brennan;Robert C.J. Wills;C. Proistosescu;C. Proistosescu
影响因子:
5.2
作者:
Lutsko, Nicholas J.;Popp, Max
通讯作者:
Popp, Max
DOI:
--
发表时间:
2012
期刊:
影响因子:
--
作者:
T. Andrews;M. Ringer;M. Doutriaux;M. Webb;W. Collins
通讯作者:
W. Collins
影响因子:
4.9
作者:
M. Lague;G. Bonan;A. Swann
通讯作者:
A. Swann