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Study of mechanism and condition for ischemic preconditioning induced by volatile anesthetics pretreatment in the liver

Study of mechanism and condition for ischemic preconditioning induced by volatile anesthetics pretreatment in the liver
挥发性麻醉药预处理引起肝脏缺血预适应的机制和条件研究
批准号：
12470314
负责人：
INABA Hideo
金额：
$6.02万
依托单位：
Kanazawa University
依托单位国家：
日本
项目类别：
Grant-in-Aid for Scientific Research (B)
财政年份：
2000
资助国家：
日本
项目状态：
已结题
起止时间：
2000 至 2002
来源：
https://kaken.nii.ac.jp/en/grant/KAKENHI-PROJECT-12470314/
关键词：
volatile anestheticsischemic preconditioningisolated perfused liverliver slice incubationapoptosispatch clampK-ATP channel虚血耐性ATP感受性Kチャンネル肝臓β細胞K(ATP)チャンネルischemic preconditioning摘出臓器灌流内皮細胞肺

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英文摘要
Halothane, isoflurane and sevoflurane attenuated ischemia-reperfusion injury in the perfused liver of fasted rats, suggesting a possible involvement of ischemic preconditioning.In the rat liver slice incubation, isoflurane attenuated apoptosis induced by hypoxic challenge. Suppression of apoptosis is predominant in the hepatocyte.Direct effect of isoflurane on K-ATP channel was studied in isolated pancreatic β cells using whole cell patch clamp method. Isoflurane suppressed K-ATP channel in a dose-dependent manner. Repeated treatment of isoflurane enhanced the suppression of K-ATP channel.
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