Assessment of role of Kir6.1 subunit (ATP-sensitive K+ channel) in J wave syndrome
Assessment of role of Kir6.1 subunit (ATP-sensitive K+ channel) in J wave syndrome
批准号:
26460334
负责人:
NAKAYA Haruaki
金额:
$3.24万
依托单位:
依托单位国家:
日本
项目类别:
Grant-in-Aid for Scientific Research (C)
财政年份:
2014
资助国家:
日本
项目状态:
已结题
起止时间:
2014-04-01 至 2017-03-31
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NOによるマウス腸内細菌叢の安定化機構
NO对小鼠肠道菌群的稳定机制
DOI:
--
发表时间:
2016
期刊:
影响因子:
--
作者:
[松本明郎, 清水建, 霊園良恵, 立花知子, 渡部恭大, 野田公俊, 中谷晴昭]
通讯作者:
中谷晴昭
循環器急性診療 Critical Care Cardiology
重症监护心脏病学
DOI:
--
发表时间:
2015
期刊:
影响因子:
--
作者:
[松本明郎, 霊園良恵, 立花知子, 清水健, 渡部恭大, 野田公俊, 中谷晴昭, 中谷晴昭]
通讯作者:
中谷晴昭
マウス腸管内におけるNO産生の可視化による殺菌機構の解析
通过观察小鼠肠道中NO的产生来分析杀菌机制
DOI:
--
发表时间:
2015
期刊:
影响因子:
--
作者:
[松本明郎, 霊園良恵, 立花知子, 清水健, 渡部恭大, 野田公俊, 中谷晴昭]
通讯作者:
中谷晴昭
Experimental assessment of effects of antiproliferative drugs of drug-eluting stents on endothelial cells
药物洗脱支架抗增殖药物对内皮细胞影响的实验评估
DOI:
10.1016/j.carrev.2015.07.002
发表时间:
2015
期刊:
Cardiovascular Revascularization Medicine
影响因子:
1.7
作者:
[Miura K, Nakaya H, Kobayashi Y.]
通讯作者:
Kobayashi Y.
Effects of selective KAch channel blocker NTC-801 on atreal fibrillation in a canine model of atrial tachpacing: comparison with class Ic and III drugs.
选择性 KAch 通道阻滞剂 NTC-801 对心房快速起搏犬模型中心房颤动的影响:与 Ic 类和 III 类药物的比较。
DOI:
10.1097/fjc.0000000000000065
发表时间:
2014
期刊:
J Cardiovasc Pharmacol
影响因子:
--
作者:
[Yamamoto W, Hashimoto N, Matsuura J, Machida T, Ogino Y, Kobayashi T, Yamanaka Y, Ishiwata N, Yamashita Y, Tanimoto K, Miyoshi S, Fukuda K, Nakaya H, Ogawa S.]
通讯作者:
Ogawa S.
共 6 条
Functional role of ATP-sensitive K^+ channel in vascular endothelial cells
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批准号:20590249
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项目类别:Grant-in-Aid for Scientific Research (C)
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资助金额:$3.08万
-
财政年份:2008
-
负责人:NAKAYA Haruaki
-
依托单位:
Molecular and functional analysis of ATP-sensitive K^+ channel on the nuclear envelope
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批准号:18590232
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项目类别:Grant-in-Aid for Scientific Research (C)
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资助金额:$2.57万
-
财政年份:2006
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负责人:NAKAYA Haruaki
-
依托单位:
Role of Kir6.1 channels in cardiomyocytes clarified by Kir6.1-transgenic mice
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批准号:15390078
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项目类别:Grant-in-Aid for Scientific Research (B)
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资助金额:$9.54万
-
财政年份:2003
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负责人:NAKAYA Haruaki
-
依托单位:
Cellular mechanisms of cardioprotection by ischemic preconditioning : Functional study using Kir6.2- (Kir6.2^<-/->) and Kir6.1-deficient (Kir6.1^<-/->) mice
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批准号:13670080
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项目类别:Grant-in-Aid for Scientific Research (C)
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资助金额:$2.62万
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财政年份:2001
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负责人:NAKAYA Haruaki
-
依托单位:
Role of cardiac ATP-sensitive K^+ channels clarified by Kir6.2-deficient mice
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批准号:11670081
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项目类别:Grant-in-Aid for Scientific Research (C)
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资助金额:$2.24万
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财政年份:1999
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负责人:NAKAYA Haruaki
-
依托单位:
Electropharmacological study of receptor-mediated regulation of cardiac Na^+-activated K^+ channels
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批准号:08670102
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项目类别:Grant-in-Aid for Scientific Research (C)
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资助金额:$1.41万
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财政年份:1996
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负责人:NAKAYA Haruaki
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依托单位:
Search for a cardiac Cl^- channel blocker : Development of a novel type of antiarrhythmic drug
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批准号:07557173
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项目类别:Grant-in-Aid for Scientific Research (A)
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资助金额:$7.3万
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财政年份:1995
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负责人:NAKAYA Haruaki
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依托单位:
Pathophysiological gignificance of endothelin receptor-mediated regulation of the cardiac ATP-sensitive K channel
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批准号:06670099
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项目类别:Grant-in-Aid for General Scientific Research (C)
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资助金额:$1.28万
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财政年份:1994
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负责人:NAKAYA Haruaki
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依托单位:
Transmembrane Cl^- Movement in Cardiac Cells and Its Pathophysiological Significance
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批准号:03670086
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项目类别:Grant-in-Aid for General Scientific Research (C)
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资助金额:$1.28万
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财政年份:1991
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负责人:NAKAYA Haruaki
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依托单位:
Possible Involvement of Increased Outward K^+ Current Induced by Intracellular Metabolic Derangement in Extracellular K^+ Accumulation during Myocardial Ischemia.
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批准号:63570085
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项目类别:Grant-in-Aid for General Scientific Research (C)
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资助金额:$1.34万
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财政年份:1988
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负责人:NAKAYA Haruaki
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依托单位:
海外基金