DRUG USE--GENERATIONAL TRANSMISSION IN MINORITY YOUTH

该项目的目的是研究三个传输 几代儿童吸毒/滥用药物的前兆。 这 纵向研究始于1990年（T1），当时第一代（G1）母亲 和第二代（G2）青少年进行了评估。 数据 文化和家庭背景，父母养育子女， 个性/行为变量。 五年后（T2），G2 对青少年进行了重新评估。 在T2数据收集时，原始 重复T1测量，并从两个样本中收集额外数据。 母亲和孩子的文化影响，个性/行为，父母- 儿童关系，同伴吸毒和自我吸毒。 T1-T2数据 确定青少年因素确实会影响以后的药物使用;我们现在 我建议通过研究G3来进一步研究这些影响 G2原始研究青少年的后代。G3儿童将在 在7-12岁时评估儿童后期药物前体 使用/滥用药物。 然后我们将追踪 途径（T1-T3），以确定药物倾向的特点是如何 传来 假设的路径，基于我们的家庭 干预模式，即文化因素和父母个性 风险特征（例如，非常规性、情绪控制不良） 导致父子相互分离（例如，少一点温暖，多一点 冲突）。 这就导致了儿童的发展风险， 药物使用/药物滥用。 我们假设这条通路， 在G1父代和G2子代中，将对G2子代重复（现在 父母）和他/她的后代。 同样有趣的是我们的考试 风险（药物有益）和保护（非药物有益） 两代人之间的互动影响G3儿童的结局。 的 研究的样本量为325名7-12岁的儿童及其父母。 T3 将使用自我报告收集数据。 分析技术将 包括结构方程和分层回归。的 研究的意义在于它描绘了代际 与儿童期毒品前体形成有关的因素 使用/滥用药物。 (By专注于如何改善心理社会 在危险的幼儿环境中，人们不仅可以减少 以后使用毒品的可能性，而且还会影响“打破常规”的变化 （三）“代际风险传递链”。

The purpose of this project is to study the transmission across three generations of the childhood precursors of drug use/drug abuse. This longitudinal study began in 1990 (T1) when first generation (G1) mothers and second generation (G2) adolescents were assessed. Data were obtained on cultural and family context, parent child-rearing, and personality/behavioral variables. Five years later (T2), the G2 adolescents were reassessed. At the T2 data collection, the original T1 measures were repeated, and additional data were gathered from both mother and child on cultural influences, personality/behavior, parent- child relations, peer drug use and self drug use. The T1-T2 data established that adolescent factors do influence later drug use; we now propose to further investigate these influences by studying the G3 offspring of the G2 original study adolescents. The G3 child will be evaluated at age 7-12 in terms of childhood precursors of later drug use/drug abuse. We will then trace the intra- and intergenerational pathways (T1-T3) to determine how drug-prone characteristics are transmitted. The hypothesized pathway, based on our Family Interactional Model, is that cultural factors and parent personality risk characteristics (e.g. unconventionality, poor control of emotions) lead to parent-child mutual detachment (e.g., less warmth, more conflict). This then leads to the development in the child of risks for drug use/drug abuse. We hypothesize that this pathway, already shown in the G1 parent and G2 child, will be repeated for the G2 child (now a parent) and his/her offspring. Also of interest is our examination of risk (drug-conducive) and protective (nondrug-conducive) intra- and intergenerational interactions as they affect G3 child outcome. The sample size for the study is 325 7-12-year-olds and their parents. T3 data will be collected using self-report. The analytic techniques will include structural equation and hierarchical regression. The significance of the study lies in its delineation of generational factors implicated in the development of childhood precursors of drug use/drug abuse. (By focusing on ways to improve the psychosocial environment of the young child at risk, one can not only lesser the likelihood of later drug use, but also effect changes that "break the chain" of generational risk transmission.)