The Role of Myc in ChREBP-dependent, Glucose-stimulated Gene Expression

Myc 在 ChREBP 依赖性、葡萄糖刺激的基因表达中的作用

• 批准号: 8147943
• 负责人: DONALD K. SCOTT
• 金额: $ 18.27万
• 依托单位: UNIVERSITY OF PITTSBURGH AT PITTSBURGH
• 依托单位国家: 美国
• 财政年份: 2003
• 资助国家: 美国
• 起止时间: 2003-07-01 至 2011-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8147943
• 关键词: Affect; Binding; Binding Proteins; Biological Assay; Carbohydrates; Cell Nucleus; Cells; Co-Immunoprecipitations; Complex; DNA-Directed RNA Polymerase; Data; Diabetes Mellitus; Disease; Environment; Epigenetic Process; Event; Failure; Fasting; Gene Expression; Gene Targeting; Genes; Genetic Transcription; Glucose; Glycolysis; Hepatic; Liver; Malignant Neoplasms; Measures; Mediating; Metabolic; Metabolic Control; Metabolic Diseases; Metabolic syndrome; Metabolism; Molecular; Molecular Conformation; Mus; Nucleic Acid Regulatory Sequences; Nucleosomes; Pattern; Perception; Phenotype; Physiological; Play; Polymerase; Positioning Attribute; Post-Translational Protein Processing; Proteomics; Proto-Oncogenes; Publishing; Pyruvate Kinase; Recruitment Activity; Response Elements; Reverse Transcriptase Polymerase Chain Reaction; Role; Structure of beta Cell of islet; Testing; Tissues; Transactivation; Transcriptional Activation; Warburg Effect; Work; activating transcription factor; blood glucose regulation; c-myc Genes; carbohydrate binding protein; carbohydrate receptor; cell growth regulation; chromatin immunoprecipitation; detection of nutrient; feeding; glucose metabolism; in vivo; insulin tolerance; prevent; promoter; public health relevance; response; therapy design; transcription factor